Tumor protein expression of the DNA repair gene BRCA1 and lethal prostate cancer.


Journal

Carcinogenesis
ISSN: 1460-2180
Titre abrégé: Carcinogenesis
Pays: England
ID NLM: 8008055

Informations de publication

Date de publication:
14 07 2020
Historique:
received: 16 03 2020
revised: 26 05 2020
accepted: 11 06 2020
pubmed: 20 6 2020
medline: 7 2 2021
entrez: 20 6 2020
Statut: ppublish

Résumé

DNA repair genes are commonly altered in metastatic prostate cancer, but BRCA1 mutations are rare. Preliminary studies suggest that higher tumor expression of the BRCA1 protein may be associated with worse prognosis. We undertook a prospective study among men with prostate cancer in the Health Professionals Follow-up Study and evaluated BRCA1 via immunohistochemical staining on tissue microarrays. BRCA1 was expressed in 60 of 589 tumors. Prevalence of BRCA1 positivity was 43% in the 14 men with metastases at diagnosis compared with 9% in non-metastatic tumors [difference, 33 percentage points; 95% confidence interval (CI), 7-59]. BRCA1-positive tumors had 2.16-fold higher Ki-67 proliferative indices (95% CI, 1.18-3.95), higher tumor aneuploidy as predicted from whole-transcriptome profiling, and higher Gleason scores. Among the 575 patients with non-metastatic disease at diagnosis, we evaluated the association between BRCA1 expression and development of lethal disease (metastasis or cancer-specific death, 69 events) during long-term follow-up (median, 18.3 years). A potential weak association of BRCA1 positivity with lethal disease (hazard ratio, 1.61; 95% CI, 0.82-3.15) was attenuated when adjusting for age, Gleason score and clinical stage (hazard ratio, 1.11; 95% CI, 0.54-2.29). In summary, BRCA1 protein expression is a feature of more proliferative and more aneuploid prostate tumors and is more common in metastatic disease. While not well suited as a prognostic biomarker in primary prostate cancer, BRCA1 protein expression may be most relevant in advanced disease.

Identifiants

pubmed: 32556091
pii: 5859098
doi: 10.1093/carcin/bgaa061
pmc: PMC7359768
doi:

Substances chimiques

BRCA1 Protein 0
BRCA1 protein, human 0
Biomarkers, Tumor 0
Neoplasm Proteins 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

904-908

Subventions

Organisme : NCI NIH HHS
ID : P50 CA090381
Pays : United States
Organisme : NCI NIH HHS
ID : U01 CA167552
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NCI NIH HHS
ID : P01 CA228696
Pays : United States
Organisme : NCI NIH HHS
ID : U01 CA113913
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA136578
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA006516
Pays : United States

Informations de copyright

© The Author(s) 2020. Published by Oxford University Press. All rights reserved. For Permissions, please email: journals.permissions@oup.com.

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Auteurs

Konrad H Stopsack (KH)

Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.

Travis Gerke (T)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.
Department of Cancer Epidemiology, Moffitt Cancer Center, Tampa, FL, USA.

Piotr Zareba (P)

Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Department of Urology, McMaster University, Hamilton, ON, USA.

Andreas Pettersson (A)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.
Department of Medicine, Clinical Epidemiology Unit, Solna, Karolinska Institutet, Stockholm, Sweden.

Dipanjan Chowdhury (D)

Department of Radiation Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, MA, USA.

Ericka M Ebot (EM)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.

Richard Flavin (R)

Department of Pathology, St. James's Hospital, Dublin, Ireland.
Trinity College, Dublin, Ireland.

Stephen Finn (S)

Department of Pathology, St. James's Hospital, Dublin, Ireland.
Trinity College, Dublin, Ireland.

Philip W Kantoff (PW)

Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Meir J Stampfer (MJ)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.
Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.

Massimo Loda (M)

Department of Pathology, Cornell Medical School, New York, NY, USA.

Michelangelo Fiorentino (M)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.
Pathology Unit, Addarii Institute, S. Orsola-Malpighi Hospital, Bologna, Italy.

Lorelei A Mucci (LA)

Department of Epidemiology, Harvard T.H. Chan School of Public Health, Boston, MA, USA.
Channing Division of Network Medicine, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.

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Classifications MeSH