Amyloidosis is associated with thicker myelin and increased oligodendrogenesis in the adult mouse brain.
Alzheimer's disease
RRID:AB_10013361
RRID:AB_10806491
RRID:AB_162542
RRID:AB_2040202
RRID:AB_2236897
RRID:AB_2493179
RRID:AB_2534017
RRID:AB_2534117
RRID:AB_2535792
RRID:AB_2535794
RRID:AB_2535864
RRID:AB_2536183
RRID:AB_2617137
RRID:AB_2827931
RRID:AB_476692
RRID:AB_570666
RRID:AB_662798
RRID:AB_91939
RRID:IMSR_JAX:006148
RRID:IMSR_JAX:007669
RRID:IMSR_JAX:008169
RRID:MMRRC_034836-JAX
RRID:SCR_000441
RRID:SCR_002798
RRID:SCR_003070
RRID:SCR_011323
amyloid
dementia
myelin
oligodendrocyte
Journal
Journal of neuroscience research
ISSN: 1097-4547
Titre abrégé: J Neurosci Res
Pays: United States
ID NLM: 7600111
Informations de publication
Date de publication:
10 2020
10 2020
Historique:
received:
23
01
2020
revised:
03
05
2020
accepted:
25
05
2020
pubmed:
20
6
2020
medline:
18
9
2021
entrez:
20
6
2020
Statut:
ppublish
Résumé
In Alzheimer's disease, amyloid plaque formation is associated with the focal death of oligodendrocytes and soluble amyloid β impairs the survival of oligodendrocytes in vitro. However, the response of oligodendrocyte progenitor cells (OPCs) to early amyloid pathology remains unclear. To explore this, we performed a histological, electrophysiological, and behavioral characterization of transgenic mice expressing a pathological form of human amyloid precursor protein (APP), containing three single point mutations associated with the development of familial Alzheimer's disease (PDGFB-APP
Identifiants
pubmed: 32557778
doi: 10.1002/jnr.24672
pmc: PMC7540704
doi:
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1905-1932Informations de copyright
© 2020 The Authors. Journal of Neuroscience Research published by Wiley Periodicals LLC.
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