The autoimmune susceptibility gene,
Animals
Bacterial Adhesion
Escherichia coli
/ genetics
Escherichia coli Infections
/ genetics
Female
Gastrointestinal Microbiome
Genetic Predisposition to Disease
Humans
Inflammatory Bowel Diseases
/ genetics
Male
Mice
Mice, Inbred BALB C
Mice, Inbred C57BL
Mice, Knockout
Protein Tyrosine Phosphatase, Non-Receptor Type 2
/ genetics
AIEC
Caco-2
LF82
colitis
epithelial
inflammatory bowel disease
macrophage
microbiome
Journal
Gut microbes
ISSN: 1949-0984
Titre abrégé: Gut Microbes
Pays: United States
ID NLM: 101495343
Informations de publication
Date de publication:
01 11 2020
01 11 2020
Historique:
pubmed:
27
6
2020
medline:
14
9
2021
entrez:
27
6
2020
Statut:
ppublish
Résumé
Inflammatory bowel disease (IBD) pathogenesis involves significant contributions from genetic and environmental factors. Loss-of-function single-nucleotide polymorphisms (SNPs) in the protein tyrosine phosphatase non-receptor type 2 (
Identifiants
pubmed: 32586195
doi: 10.1080/19490976.2020.1775538
pmc: PMC7524159
doi:
Substances chimiques
Protein Tyrosine Phosphatase, Non-Receptor Type 2
EC 3.1.3.48
Ptpn2 protein, mouse
EC 3.1.3.48
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1547-1566Subventions
Organisme : NIDDK NIH HHS
ID : R01 DK091281
Pays : United States
Organisme : NIGMS NIH HHS
ID : R35 GM124724
Pays : United States
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