Risk Factors of Cerebellar Microembolic Infarctions After Carotid Artery Stenting.


Journal

World neurosurgery
ISSN: 1878-8769
Titre abrégé: World Neurosurg
Pays: United States
ID NLM: 101528275

Informations de publication

Date de publication:
10 2020
Historique:
received: 05 05 2020
revised: 24 06 2020
accepted: 25 06 2020
pubmed: 6 7 2020
medline: 31 3 2021
entrez: 5 7 2020
Statut: ppublish

Résumé

This study analyzes the incidence of microembolic infarctions (MIs) in the cerebellum after carotid artery stenting (CAS) to determine the risk factors. From 2012 to 2019, 162 CASs in 155 patients were performed at our hospital. Fifty-seven patients (35.7%) showing new MIs on diffusion-weighted imaging after CAS were enrolled. Patients were assigned to either the cerebellar group (n = 14, 8.8%) if their MIs were in the cerebellum and/or cerebrum or the cerebral group (n = 43, 26.9%) if their MIs were only in the cerebrum. Patient characteristics, anatomic features, and clinical data were retrospectively compared between the 2 groups. Advanced age, right-sided carotid stenosis, severe calcification of aortic arch and brachiocephalic trunk, and vertebral artery narrowing with intraprocedural hemodynamic depression (IHD) significantly increased the development of cerebellar MIs. On multivariate analysis, advanced age, right-sided carotid stenosis, and vertebral artery narrowing with IHD were independent predictors of developing new cerebellar MIs. Cerebellar MIs after CAS were not uncommon. Catheter maneuvering in the aortic arch or the brachiocephalic trunk could be the main cause of thromboemboli in cerebellar MIs. Careful attention should be paid to catheter maneuvering, especially in older patients with right-sided carotid lesions. In addition, cerebellar hypoperfusion caused by vertebral artery narrowing with IHD might reduce washout of debris, a cause of cerebellar MIs.

Sections du résumé

BACKGROUND
This study analyzes the incidence of microembolic infarctions (MIs) in the cerebellum after carotid artery stenting (CAS) to determine the risk factors.
METHODS
From 2012 to 2019, 162 CASs in 155 patients were performed at our hospital. Fifty-seven patients (35.7%) showing new MIs on diffusion-weighted imaging after CAS were enrolled. Patients were assigned to either the cerebellar group (n = 14, 8.8%) if their MIs were in the cerebellum and/or cerebrum or the cerebral group (n = 43, 26.9%) if their MIs were only in the cerebrum. Patient characteristics, anatomic features, and clinical data were retrospectively compared between the 2 groups.
RESULTS
Advanced age, right-sided carotid stenosis, severe calcification of aortic arch and brachiocephalic trunk, and vertebral artery narrowing with intraprocedural hemodynamic depression (IHD) significantly increased the development of cerebellar MIs. On multivariate analysis, advanced age, right-sided carotid stenosis, and vertebral artery narrowing with IHD were independent predictors of developing new cerebellar MIs. Cerebellar MIs after CAS were not uncommon.
CONCLUSIONS
Catheter maneuvering in the aortic arch or the brachiocephalic trunk could be the main cause of thromboemboli in cerebellar MIs. Careful attention should be paid to catheter maneuvering, especially in older patients with right-sided carotid lesions. In addition, cerebellar hypoperfusion caused by vertebral artery narrowing with IHD might reduce washout of debris, a cause of cerebellar MIs.

Identifiants

pubmed: 32622063
pii: S1878-8750(20)31475-3
doi: 10.1016/j.wneu.2020.06.207
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e290-e296

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Auteurs

Hidemichi Ito (H)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan. Electronic address: hdmcito@marianna-u.ac.jp.

Masashi Uchida (M)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Tomohiro Kaji (T)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Yuki Go (Y)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Gaku Hidaka (G)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Hiroshi Takasuna (H)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Tetsuya Goto (T)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Ichiro Takumi (I)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

Yuichiro Tanaka (Y)

Department of Neurosurgery, St. Marianna University School of Medicine, Miyamae-ku, Kawasaki, Kanagawa, Japan.

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