Intergenerational transmission of childhood trauma? Testing cellular aging in mothers exposed to sexual abuse and their children.


Journal

Psychoneuroendocrinology
ISSN: 1873-3360
Titre abrégé: Psychoneuroendocrinology
Pays: England
ID NLM: 7612148

Informations de publication

Date de publication:
10 2020
Historique:
received: 13 02 2020
revised: 10 06 2020
accepted: 19 06 2020
pubmed: 7 7 2020
medline: 20 7 2021
entrez: 7 7 2020
Statut: ppublish

Résumé

Exposure to maltreatment in childhood can lead to increased risk for poor health outcomes in adulthood. Child maltreatment and later poor health may be linked by premature biological aging. We tested whether childhood sexual abuse (CSA) was associated with telomere length (TL) in adult females. We further tested the hypothesis of intergenerational transmission of CSA-related effects by measuring TL in both CSA-exposed and non-exposed mothers and their children. Participants were a subset of females and their children in a prospective-longitudinal cohort study of sexually abused females and a demographically comparable control group from the same Washington, D.C. area. TL was measured using qPCR in both leukocyte and buccal samples from females (N = 108, mean age 36.3 years) and buccal samples from their children (N = 124, mean age 10.5 years). Multilevel models were used to test associations between CSA-exposure and TL measured in leukocytes and buccal tissue in females and to test the intergenerational effect of maternal-CSA exposure on age-adjusted TL in their children. CSA-exposure was not associated with TL in adult females. Maternal TL and biological sex were significant predictors of child TL such that longer maternal TL predicted longer TL in children, and female children had longer TL than male children. However, maternal-CSA exposure did not predict TL in children. CSA-exposure was not associated with TL in this cohort of middle-aged females, nor was there evidence for an intergenerational effect of maternal-CSA exposure on child TL. This finding is in line with some previous results on CSA and adult TL. Previous significant results associating child maltreatment with shorter TL may be capturing a population of individuals exposed to either multiple types of maltreatment compared to controls with no childhood adversity, or maltreatment in childhood with concurrent TL measurements.

Sections du résumé

BACKGROUND
Exposure to maltreatment in childhood can lead to increased risk for poor health outcomes in adulthood. Child maltreatment and later poor health may be linked by premature biological aging. We tested whether childhood sexual abuse (CSA) was associated with telomere length (TL) in adult females. We further tested the hypothesis of intergenerational transmission of CSA-related effects by measuring TL in both CSA-exposed and non-exposed mothers and their children.
METHODS
Participants were a subset of females and their children in a prospective-longitudinal cohort study of sexually abused females and a demographically comparable control group from the same Washington, D.C. area. TL was measured using qPCR in both leukocyte and buccal samples from females (N = 108, mean age 36.3 years) and buccal samples from their children (N = 124, mean age 10.5 years). Multilevel models were used to test associations between CSA-exposure and TL measured in leukocytes and buccal tissue in females and to test the intergenerational effect of maternal-CSA exposure on age-adjusted TL in their children.
RESULTS
CSA-exposure was not associated with TL in adult females. Maternal TL and biological sex were significant predictors of child TL such that longer maternal TL predicted longer TL in children, and female children had longer TL than male children. However, maternal-CSA exposure did not predict TL in children.
DISCUSSION
CSA-exposure was not associated with TL in this cohort of middle-aged females, nor was there evidence for an intergenerational effect of maternal-CSA exposure on child TL. This finding is in line with some previous results on CSA and adult TL. Previous significant results associating child maltreatment with shorter TL may be capturing a population of individuals exposed to either multiple types of maltreatment compared to controls with no childhood adversity, or maltreatment in childhood with concurrent TL measurements.

Identifiants

pubmed: 32629221
pii: S0306-4530(20)30202-X
doi: 10.1016/j.psyneuen.2020.104781
pmc: PMC7502488
mid: NIHMS1605503
pii:
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

104781

Subventions

Organisme : NIEHS NIH HHS
ID : U01 ES030949
Pays : United States
Organisme : NICHD NIH HHS
ID : P50 HD089922
Pays : United States
Organisme : NICHD NIH HHS
ID : R01 HD072468
Pays : United States
Organisme : NIA NIH HHS
ID : R21 AG055621
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG048791
Pays : United States
Organisme : NIA NIH HHS
ID : T32 AG049676
Pays : United States

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

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Auteurs

Laura Etzel (L)

Department of Biobehavioral Health, The Pennsylvania State University, University Park, PA, USA.

Waylon J Hastings (WJ)

Department of Biobehavioral Health, The Pennsylvania State University, University Park, PA, USA.

Brooke C Mattern (BC)

Department of Biobehavioral Health, The Pennsylvania State University, University Park, PA, USA.

Monica L Oxford (ML)

Department of Family and Child Nursing, University of Washington, Seattle, WA, USA.

Christine Heim (C)

Department of Biobehavioral Health, The Pennsylvania State University, University Park, PA, USA; Charité - Universitätsmedizin Berlin, Corporate Member of Freie Universität Berlin, Humboldt-Universität zu Berlin, Berlin Institute of Health (BIH), Institute of Medical Psychology, Berlin, Germany.

Frank W Putnam (FW)

Department of Psychiatry, University of North Carolina School of Medicine, Chapel Hill, NC, USA.

Jennie G Noll (JG)

Department of Human Development and Family Studies, The Pennsylvania State University, University Park, PA, USA.

Idan Shalev (I)

Department of Biobehavioral Health, The Pennsylvania State University, University Park, PA, USA. Electronic address: ius14@psu.edu.

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