Inhibition of impulsive action by projection-defined prefrontal pyramidal neurons.


Journal

Proceedings of the National Academy of Sciences of the United States of America
ISSN: 1091-6490
Titre abrégé: Proc Natl Acad Sci U S A
Pays: United States
ID NLM: 7505876

Informations de publication

Date de publication:
21 07 2020
Historique:
pubmed: 8 7 2020
medline: 15 9 2020
entrez: 8 7 2020
Statut: ppublish

Résumé

The prefrontal cortex (PFC) plays a critical role in curbing impulsive behavior, but the underlying circuit mechanism remains incompletely understood. Here we show that a subset of dorsomedial PFC (dmPFC) layer 5 pyramidal neurons, which project to the subthalamic nucleus (STN) of the basal ganglia, play a key role in inhibiting impulsive responses in a go/no-go task. Projection-specific labeling and calcium imaging showed that the great majority of STN-projecting neurons were preferentially active in no-go trials when the mouse successfully withheld licking responses, but lateral hypothalamus (LH)-projecting neurons were more active in go trials with licking; visual cortex (V1)-projecting neurons showed only weak task-related activity. Optogenetic activation and inactivation of STN-projecting neurons reduced and increased inappropriate licking, respectively, partly through their direct innervation of the STN, but manipulating LH-projecting neurons had the opposite effects. These results identify a projection-defined subtype of PFC pyramidal neurons as key mediators of impulse control.

Identifiants

pubmed: 32631999
pii: 2000523117
doi: 10.1073/pnas.2000523117
pmc: PMC7382266
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

17278-17287

Subventions

Organisme : Howard Hughes Medical Institute
Pays : United States

Déclaration de conflit d'intérêts

The authors declare no competing interest.

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Auteurs

Bing Li (B)

Division of Neurobiology, Department of Molecular and Cell Biology, Helen Wills Neuroscience Institute, University of California, Berkeley, CA 94720.
Howard Hughes Medical Institute, University of California, Berkeley, CA 94720.

Thao Phuong Nguyen (TP)

Bioengineering and Materials Science Engineering, University of California, Berkeley, CA 94720.

Chenyan Ma (C)

Division of Neurobiology, Department of Molecular and Cell Biology, Helen Wills Neuroscience Institute, University of California, Berkeley, CA 94720.
Howard Hughes Medical Institute, University of California, Berkeley, CA 94720.

Yang Dan (Y)

Division of Neurobiology, Department of Molecular and Cell Biology, Helen Wills Neuroscience Institute, University of California, Berkeley, CA 94720; ydan@berkeley.edu.
Howard Hughes Medical Institute, University of California, Berkeley, CA 94720.

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