Adipocyte Fatty Acid Transfer Supports Megakaryocyte Maturation.


Journal

Cell reports
ISSN: 2211-1247
Titre abrégé: Cell Rep
Pays: United States
ID NLM: 101573691

Informations de publication

Date de publication:
07 07 2020
Historique:
received: 11 09 2019
revised: 21 02 2020
accepted: 15 06 2020
entrez: 9 7 2020
pubmed: 9 7 2020
medline: 29 4 2021
Statut: ppublish

Résumé

Megakaryocytes (MKs) come from a complex process of hematopoietic progenitor maturation within the bone marrow that gives rise to de novo circulating platelets. Bone marrow microenvironment contains a large number of adipocytes with a still ill-defined role. This study aims to analyze the influence of adipocytes and increased medullar adiposity in megakaryopoiesis. An in vivo increased medullar adiposity in mice caused by high-fat-diet-induced obesity is associated to an enhanced MK maturation and proplatelet formation. In vitro co-culture of adipocytes with bone marrow hematopoietic progenitors shows that delipidation of adipocytes directly supports MK maturation by enhancing polyploidization, amplifying the demarcation membrane system, and accelerating proplatelet formation. This direct crosstalk between adipocytes and MKs occurs through adipocyte fatty acid transfer to MKs involving CD36 to reinforce megakaryocytic maturation. Thus, these findings unveil an influence of adiposity on MK homeostasis based on a dialogue between adipocytes and MKs.

Identifiants

pubmed: 32640240
pii: S2211-1247(20)30856-1
doi: 10.1016/j.celrep.2020.107875
pii:
doi:

Substances chimiques

CD36 Antigens 0
Fatty Acids 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

107875

Informations de copyright

Copyright © 2020 The Author(s). Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Interests The authors declare no competing interests.

Auteurs

Colin Valet (C)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Aurelie Batut (A)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Alicia Vauclard (A)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Alizee Dortignac (A)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Marie Bellio (M)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Bernard Payrastre (B)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France; Hematology Laboratory, Toulouse University Hospital, Toulouse, France.

Philippe Valet (P)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France.

Sonia Severin (S)

INSERM U1048 and Paul Sabatier University, Institute of Cardiovascular and Metabolic Diseases, Toulouse, France. Electronic address: sonia.severin@inserm.fr.

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Classifications MeSH