Limonene, a food additive, and its active metabolite perillyl alcohol improve regeneration and attenuate neuropathic pain after peripheral nerve injury: Evidence for IL-1β, TNF-α, GAP, NGF and ERK involvement.


Journal

International immunopharmacology
ISSN: 1878-1705
Titre abrégé: Int Immunopharmacol
Pays: Netherlands
ID NLM: 100965259

Informations de publication

Date de publication:
Sep 2020
Historique:
received: 18 05 2020
revised: 25 06 2020
accepted: 29 06 2020
pubmed: 12 7 2020
medline: 28 5 2021
entrez: 12 7 2020
Statut: ppublish

Résumé

Limonene (LIM) and its main metabolite perillyl alcohol (POH) are ingredients found in food with promising chemical entities due to their pharmacological profile. In this study, we hypothesized that LIM and POH are two molecules capable of accelerating the regenerative process and alleviating neuropathic pain. Animals were treated daily (LIM, POH and saline) for 28 days and during this period evaluated for mechanical hyperalgesia, astrocyte participation by immunofluorescence for GFAP, and ELISA was used to quantify IL-1β and TNF-α in the spinal cord. Western blot analysis of the following proteins was also performed: GFAP, GAP-43, NGF and ERK. For motor deficit analysis, tests were performed to assess hind paw muscle strength and footprints through gait (SFI). Both POH and LIM accelerated the regenerative process and improved motor deficits comparing to positive control; however, POH was more effective, particularly between the 2nd and 3rd week after the nerve injury, increasing GAP-43, NGF and the phosphorylated ERK immunocontent. Moreover, POH and LIM were able to reduce hyperalgesia and astrocytosis. Both substances, LIM and POH, improved the regeneration process and sensory and motor function recovery in the PNI model in mice by mitigating the inflammatory reactions and up-regulating the neurotrophic process.

Sections du résumé

BACKGROUND BACKGROUND
Limonene (LIM) and its main metabolite perillyl alcohol (POH) are ingredients found in food with promising chemical entities due to their pharmacological profile. In this study, we hypothesized that LIM and POH are two molecules capable of accelerating the regenerative process and alleviating neuropathic pain.
METHODS METHODS
Animals were treated daily (LIM, POH and saline) for 28 days and during this period evaluated for mechanical hyperalgesia, astrocyte participation by immunofluorescence for GFAP, and ELISA was used to quantify IL-1β and TNF-α in the spinal cord. Western blot analysis of the following proteins was also performed: GFAP, GAP-43, NGF and ERK. For motor deficit analysis, tests were performed to assess hind paw muscle strength and footprints through gait (SFI).
RESULTS RESULTS
Both POH and LIM accelerated the regenerative process and improved motor deficits comparing to positive control; however, POH was more effective, particularly between the 2nd and 3rd week after the nerve injury, increasing GAP-43, NGF and the phosphorylated ERK immunocontent. Moreover, POH and LIM were able to reduce hyperalgesia and astrocytosis.
CONCLUSIONS CONCLUSIONS
Both substances, LIM and POH, improved the regeneration process and sensory and motor function recovery in the PNI model in mice by mitigating the inflammatory reactions and up-regulating the neurotrophic process.

Identifiants

pubmed: 32652504
pii: S1567-5769(20)31617-9
doi: 10.1016/j.intimp.2020.106766
pii:
doi:

Substances chimiques

Anti-Inflammatory Agents 0
Food Additives 0
GTPase-Activating Proteins 0
Interleukin-1beta 0
Monoterpenes 0
Tumor Necrosis Factor-alpha 0
perillyl alcohol 319R5C7293
Nerve Growth Factor 9061-61-4
Limonene 9MC3I34447
Extracellular Signal-Regulated MAP Kinases EC 2.7.11.24

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

106766

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Heitor G Araújo-Filho (HG)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Erik W M Pereira (EWM)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Luana Heimfarth (L)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Brenda Souza Monteiro (B)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Fabiolla Rocha Santos Passos (FR)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Pollyana Siqueira-Lima (P)

Franco-Brazilian Network on Natural Products (FB2NP), Brazil; College of Science and Technology, Feira de Santana, BA 44000-000, Brazil.

Sathiyabama R Gandhi (SR)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Márcio Roberto Viana Dos Santos (MR)

Franco-Brazilian Network on Natural Products (FB2NP), Brazil; Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil.

Jackson Roberto Guedes da Silva Almeida (JR)

Franco-Brazilian Network on Natural Products (FB2NP), Brazil; Colegiado de Farmácia, Universidade Federal do Vale do São Francisco, Petrolina, PE 56304-917, Brazil.

Laurent Picot (L)

Franco-Brazilian Network on Natural Products (FB2NP), Brazil; La Rochelle Université, UMRi CNRS 7266 LIENSs, 17042 La Rochelle, France.

Raphaël Grougnet (R)

Franco-Brazilian Network on Natural Products (FB2NP), Brazil; UMR CNRS 8038, Faculty of Pharmacy, University of Paris, France.

Ray S Almeida (RS)

Department of Biological Chemistry, Regional University of Cariri, Crato, CE 63105-000, Brazil.

Henrique Douglas Melo Coutinho (H)

Department of Biological Chemistry, Regional University of Cariri, Crato, CE 63105-000, Brazil.

Lucindo J Quintans-Júnior (LJ)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil.

Natália Martins (N)

Faculty of Medicine, University of Porto, Alameda Prof. Hernâni Monteiro, 4200-319 Porto, Portugal; Institute for Research and Innovation in Health, Rua Alfredo Allen, 4200-135 Porto, Portugal. Electronic address: ncmartins@med.up.pt.

Jullyana S S Quintans (JSS)

Laboratory of Neuroscience and Pharmacological Assays (LANEF), Department of Physiology, Federal University of Sergipe, São Cristóvão, SE 49100-000, Brazil; Franco-Brazilian Network on Natural Products (FB2NP), Brazil. Electronic address: jullyanaquintans@gmail.com.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH