Determination of immunophenotypic aberrancies provides better assessment of peripheral blood involvement by mycosis fungoides/Sézary syndrome than quantification of CD26- or CD7- CD4+ T-cells.


Journal

Cytometry. Part B, Clinical cytometry
ISSN: 1552-4957
Titre abrégé: Cytometry B Clin Cytom
Pays: United States
ID NLM: 101235690

Informations de publication

Date de publication:
03 2021
Historique:
revised: 12 05 2020
received: 18 03 2020
accepted: 10 06 2020
pubmed: 16 7 2020
medline: 15 12 2021
entrez: 16 7 2020
Statut: ppublish

Résumé

Blood involvement by mycosis fungoides (MF)/Sézary syndrome (SS) influences prognosis and therapeutic decisions. MF/SS blood stage is currently determined by absolute CD4 + CD26- or CD4 + CD7-cell counts, which quantification method may overestimate MF/SS by including CD26- or CD7- normal CD4+ T-cells, or underestimate disease burden when MF/SS cells show incomplete loss of CD26 and/or CD7. Recently, through the standardization effort led by the International Clinical Cytometry Society (ICCS), recommendation was made to quantify MF/SS by enumerating immunophenotypically aberrant CD4+ T-cells, rather than CD26- or CD7- in isolation. We compared these two quantitation methods in 309 MF/SS patients who had blood samples analyzed by flow cytometry immunophenotyping (FCI) over a 1-year period. Using the European Organization of Research and Treatment of Cancer (EORTC)/International Society for Cutaneous Lymphomas (ISCL) criteria, 221 (71.5%) patients had a blood stage corresponding to B0, 57 (18.4%) to B1, and 31 (10%) to B2. By FCI analysis, a total of 62 patients (20.0%) were found positive for MF/SS. Among EORTC B0 patients, 11/221 (5%) were positive by FCI (false negatives), and among EORTC Stage B1 patients, 35/57 (61%) were negative by FCI (false positives). Regarding patients positive for MF/SS cells by FCI, there was an overall excellent correlation (r = .999, p < .001) between the EORTC/ISCL method and FCI method; however, four (6.5%) patients would have an altered B stage between B0 and B1. The MF/SS cell quantification method using immunophenotypic aberrancies, as recommended by the ICCS, allows to distinguish MF/SS cells from background benign T-cells and enables for more accurate staging, especially among patients currently being considered to have B0 and B1 stage diseases.

Sections du résumé

BACKGROUND
Blood involvement by mycosis fungoides (MF)/Sézary syndrome (SS) influences prognosis and therapeutic decisions. MF/SS blood stage is currently determined by absolute CD4 + CD26- or CD4 + CD7-cell counts, which quantification method may overestimate MF/SS by including CD26- or CD7- normal CD4+ T-cells, or underestimate disease burden when MF/SS cells show incomplete loss of CD26 and/or CD7. Recently, through the standardization effort led by the International Clinical Cytometry Society (ICCS), recommendation was made to quantify MF/SS by enumerating immunophenotypically aberrant CD4+ T-cells, rather than CD26- or CD7- in isolation.
METHODS
We compared these two quantitation methods in 309 MF/SS patients who had blood samples analyzed by flow cytometry immunophenotyping (FCI) over a 1-year period.
RESULTS
Using the European Organization of Research and Treatment of Cancer (EORTC)/International Society for Cutaneous Lymphomas (ISCL) criteria, 221 (71.5%) patients had a blood stage corresponding to B0, 57 (18.4%) to B1, and 31 (10%) to B2. By FCI analysis, a total of 62 patients (20.0%) were found positive for MF/SS. Among EORTC B0 patients, 11/221 (5%) were positive by FCI (false negatives), and among EORTC Stage B1 patients, 35/57 (61%) were negative by FCI (false positives). Regarding patients positive for MF/SS cells by FCI, there was an overall excellent correlation (r = .999, p < .001) between the EORTC/ISCL method and FCI method; however, four (6.5%) patients would have an altered B stage between B0 and B1.
CONCLUSION
The MF/SS cell quantification method using immunophenotypic aberrancies, as recommended by the ICCS, allows to distinguish MF/SS cells from background benign T-cells and enables for more accurate staging, especially among patients currently being considered to have B0 and B1 stage diseases.

Identifiants

pubmed: 32667737
doi: 10.1002/cyto.b.21933
doi:

Substances chimiques

Antigens, CD7 0
DPP4 protein, human EC 3.4.14.5
Dipeptidyl Peptidase 4 EC 3.4.14.5

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

183-191

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2020 International Clinical Cytometry Society.

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Auteurs

Kirill A Lyapichev (KA)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Ismael Bah (I)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Auris Huen (A)

Department of Dermatology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Madeleine Duvic (M)

Department of Dermatology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Mark J Routbort (MJ)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Wei Wang (W)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Jeffrey L Jorgensen (JL)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

L Jeffrey Medeiros (LJ)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Francisco Vega (F)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

Fiona E Craig (FE)

Division of Hematopathology, Mayo Clinic Arizona, Phoenix, Arizona, USA.

Sa A Wang (SA)

Department of Hematopathology, The University of Texas MD Anderson Cancer Center, Houston, Texas, USA.

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