Distinct conformational states of SARS-CoV-2 spike protein.


Journal

Science (New York, N.Y.)
ISSN: 1095-9203
Titre abrégé: Science
Pays: United States
ID NLM: 0404511

Informations de publication

Date de publication:
25 09 2020
Historique:
received: 20 06 2020
accepted: 14 07 2020
pubmed: 23 7 2020
medline: 22 10 2020
entrez: 23 7 2020
Statut: ppublish

Résumé

Intervention strategies are urgently needed to control the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pandemic. The trimeric viral spike (S) protein catalyzes fusion between viral and target cell membranes to initiate infection. Here, we report two cryo-electron microscopy structures derived from a preparation of the full-length S protein, representing its prefusion (2.9-angstrom resolution) and postfusion (3.0-angstrom resolution) conformations, respectively. The spontaneous transition to the postfusion state is independent of target cells. The prefusion trimer has three receptor-binding domains clamped down by a segment adjacent to the fusion peptide. The postfusion structure is strategically decorated by N-linked glycans, suggesting possible protective roles against host immune responses and harsh external conditions. These findings advance our understanding of SARS-CoV-2 entry and may guide the development of vaccines and therapeutics.

Identifiants

pubmed: 32694201
pii: science.abd4251
doi: 10.1126/science.abd4251
pmc: PMC7464562
doi:

Substances chimiques

Receptors, Virus 0
Spike Glycoprotein, Coronavirus 0
spike protein, SARS-CoV-2 0
Peptidyl-Dipeptidase A EC 3.4.15.1
ACE2 protein, human EC 3.4.17.23
Angiotensin-Converting Enzyme 2 EC 3.4.17.23

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1586-1592

Commentaires et corrections

Type : UpdateOf

Informations de copyright

Copyright © 2020 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works.

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Auteurs

Yongfei Cai (Y)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA.
Department of Pediatrics, Harvard Medical School, Boston, MA 02115, USA.

Jun Zhang (J)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA.
Department of Pediatrics, Harvard Medical School, Boston, MA 02115, USA.

Tianshu Xiao (T)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA.
Department of Pediatrics, Harvard Medical School, Boston, MA 02115, USA.

Hanqin Peng (H)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA.

Sarah M Sterling (SM)

The Harvard Cryo-EM Center for Structural Biology, Harvard Medical School, Boston, MA 02115, USA.
Department of Biological Chemistry and Molecular Pharmacology, Blavatnik Institute, Harvard Medical School, Boston, MA 02115, USA.

Richard M Walsh (RM)

The Harvard Cryo-EM Center for Structural Biology, Harvard Medical School, Boston, MA 02115, USA.
Department of Biological Chemistry and Molecular Pharmacology, Blavatnik Institute, Harvard Medical School, Boston, MA 02115, USA.

Shaun Rawson (S)

The Harvard Cryo-EM Center for Structural Biology, Harvard Medical School, Boston, MA 02115, USA.
Department of Biological Chemistry and Molecular Pharmacology, Blavatnik Institute, Harvard Medical School, Boston, MA 02115, USA.
SBGrid Consortium, Harvard Medical School, Boston, MA 02115, USA.

Sophia Rits-Volloch (S)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA.

Bing Chen (B)

Division of Molecular Medicine, Boston Children's Hospital, Boston, MA 02115, USA. bchen@crystal.harvard.edu.
Department of Pediatrics, Harvard Medical School, Boston, MA 02115, USA.

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Classifications MeSH