The association between plasma proteomics and incident cardiovascular disease identifies MMP-12 as a promising cardiovascular risk marker in patients with chronic kidney disease.


Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
08 2020
Historique:
received: 05 03 2020
revised: 19 05 2020
accepted: 18 06 2020
pubmed: 24 7 2020
medline: 24 6 2021
entrez: 24 7 2020
Statut: ppublish

Résumé

Previous proteomics efforts in patients with chronic kidney disease (CKD) have predominantly evaluated urinary protein levels. Therefore, our aim was to investigate the association between plasma levels of 80 cardiovascular disease-related proteins and the risk of major adverse cardiovascular events (MACE) in patients with CKD. Individuals with CKD stages 3-5 (eGFR below 60 ml min-1 [1.73 m]-2) from three community-based cohorts (PIVUS, ULSAM, SAVA), one diabetes cohort (CARDIPP) and one cohort with peripheral artery disease patients (PADVA) with information on 80 plasma protein biomarkers, assessed with a proximity extension assay, and follow-up data on incident MACE, were used as discovery sample. To validate findings and to asses generalizability to patients with CKD in clinical practice, an outpatient CKD-cohort (Malnutrition, Inflammation and Vascular Calcification (MIVC)) was used as replication sample. In the discovery sample (total n = 1316), 249 individuals experienced MACE during 7.0 ± 2.9 years (range 0.005-12.9) of follow-up, and in the replication sample, 71 MACE events in 283 individuals over a mean ± SD change of 2.9 ± 1.2 years (range 0.1-4.0) were documented. Applying Bonferroni correction, 18 proteins were significantly associated with risk of MACE in the discovery cohort, adjusting for age and sex in order of significance, GDF-15, FGF-23, REN, FABP4, IL6, TNF-R1, AGRP, MMP-12, AM, KIM-1, TRAILR2, TNFR2, CTSL1, CSF1, PlGF, CA-125, CCL20 and PAR-1 (p < 0.000625 for all). Only matrix metalloproteinase 12 (MMP-12) was significantly associated with an increased risk of MACE in the replication sample (hazard ratio (HR) per SD increase, 1.36, 95% CI (1.07-1.75), p = 0.013). Our proteomics analyses identified plasma MMP-12 as a promising cardiovascular risk marker in patients with CKD.

Sections du résumé

BACKGROUND AND AIMS
Previous proteomics efforts in patients with chronic kidney disease (CKD) have predominantly evaluated urinary protein levels. Therefore, our aim was to investigate the association between plasma levels of 80 cardiovascular disease-related proteins and the risk of major adverse cardiovascular events (MACE) in patients with CKD.
METHODS
Individuals with CKD stages 3-5 (eGFR below 60 ml min-1 [1.73 m]-2) from three community-based cohorts (PIVUS, ULSAM, SAVA), one diabetes cohort (CARDIPP) and one cohort with peripheral artery disease patients (PADVA) with information on 80 plasma protein biomarkers, assessed with a proximity extension assay, and follow-up data on incident MACE, were used as discovery sample. To validate findings and to asses generalizability to patients with CKD in clinical practice, an outpatient CKD-cohort (Malnutrition, Inflammation and Vascular Calcification (MIVC)) was used as replication sample.
RESULTS
In the discovery sample (total n = 1316), 249 individuals experienced MACE during 7.0 ± 2.9 years (range 0.005-12.9) of follow-up, and in the replication sample, 71 MACE events in 283 individuals over a mean ± SD change of 2.9 ± 1.2 years (range 0.1-4.0) were documented. Applying Bonferroni correction, 18 proteins were significantly associated with risk of MACE in the discovery cohort, adjusting for age and sex in order of significance, GDF-15, FGF-23, REN, FABP4, IL6, TNF-R1, AGRP, MMP-12, AM, KIM-1, TRAILR2, TNFR2, CTSL1, CSF1, PlGF, CA-125, CCL20 and PAR-1 (p < 0.000625 for all). Only matrix metalloproteinase 12 (MMP-12) was significantly associated with an increased risk of MACE in the replication sample (hazard ratio (HR) per SD increase, 1.36, 95% CI (1.07-1.75), p = 0.013).
CONCLUSIONS
Our proteomics analyses identified plasma MMP-12 as a promising cardiovascular risk marker in patients with CKD.

Identifiants

pubmed: 32702535
pii: S0021-9150(20)30320-8
doi: 10.1016/j.atherosclerosis.2020.06.013
pii:
doi:

Substances chimiques

Biomarkers 0
FGF23 protein, human 0
Fibroblast Growth Factor-23 7Q7P4S7RRE
MMP12 protein, human EC 3.4.24.65
Matrix Metalloproteinase 12 EC 3.4.24.65

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

11-15

Informations de copyright

Copyright © 2020 Elsevier B.V. All rights reserved.

Auteurs

Tobias Feldreich (T)

Education, Health and Social Studies, Dalarna University, Falun, Sweden. Electronic address: trf@du.se.

Christoph Nowak (C)

Division of Family Medicine and Primary Care, Department of Neurobiology, Care Sciences and Society (NVS), Karolinska Institutet, Alfred Nobels Allé 23, SE 14183, Huddinge, Sweden.

Axel C Carlsson (AC)

Division of Family Medicine and Primary Care, Department of Neurobiology, Care Sciences and Society (NVS), Karolinska Institutet, Alfred Nobels Allé 23, SE 14183, Huddinge, Sweden.

Carl-Johan Östgren (CJ)

Department of Health, Medicine and Caring Sciences, Linköping University, Linköping, Sweden.

Fredrik H Nyström (FH)

Department of Health, Medicine and Caring Sciences, Linköping University, Linköping, Sweden.

Johan Sundström (J)

Department of Medical Sciences, Uppsala University, Uppsala, Sweden.

Juan-Jesus Carrero-Roig (JJ)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.

Jerzy Leppert (J)

Centre for Clinical Research, Uppsala University, Västerås, Sweden.

Pär Hedberg (P)

Centre for Clinical Research, Uppsala University, Västerås, Sweden.

Vilmantas Giedraitis (V)

Department of Public Health and Caring Sciences, Geriatrics, Uppsala University, Uppsala, Sweden.

Lars Lind (L)

Department of Medical Sciences, Uppsala University, Uppsala, Sweden.

Antonio Cordeiro (A)

Department of Hypertension and Nephrology, Dante Pazzanese Institute of Cardiology, São Paulo, Brazil.

Johan Ärnlöv (J)

Education, Health and Social Studies, Dalarna University, Falun, Sweden; Division of Family Medicine and Primary Care, Department of Neurobiology, Care Sciences and Society (NVS), Karolinska Institutet, Alfred Nobels Allé 23, SE 14183, Huddinge, Sweden.

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Classifications MeSH