Clinical outcomes of definitive whole pelvic radiotherapy for clinical lymph node metastatic prostate cancer.


Journal

Cancer medicine
ISSN: 2045-7634
Titre abrégé: Cancer Med
Pays: United States
ID NLM: 101595310

Informations de publication

Date de publication:
09 2020
Historique:
received: 04 11 2019
revised: 21 02 2020
accepted: 23 02 2020
pubmed: 5 8 2020
medline: 21 7 2021
entrez: 5 8 2020
Statut: ppublish

Résumé

In this study, we aim to present the clinical outcomes of radiotherapy (RT) in clinical pelvic lymph node-positive prostate cancer (cN1) patients. We also analyze the prognostic factors with focus on RT dose escalation to metastatic lymph nodes (LN). We retrospectively analyzed the data from cN1 patients who were treated with definitive RT and androgen deprivation therapy (ADT) between June 2004 and February 2016. All patients received localized irradiation to the prostate region and whole pelvis irradiation. Some patients received intensity-modulated radiation therapy with RT dose escalation to metastatic LN. Univariate analyses using log-rank test were performed to find prognostic factors between patient subgroups. Fifty-one consecutive patients were identified. The median follow-up period for all patients was 88 (range 20-157) months. Primary Gleason pattern and LN RT dose were statistically significant prognostic factors for relapse-free survival (RFS) and distant metastasis-free survival (DMFS). Especially, RT dose escalation (60 Gy or more) to metastatic LN significantly improved RFS and DMFS compared with standard dose RT (4-year RFS 90.6% vs 82.1%, 7-year RFS 90.6% vs 58.0%, P = .015; 4-year DMFS 90.6% vs 82.1%, 7-year DMFS 90.6% vs 62.8%, P = .023). The following factors were all statistically significant for biochemical relapse-free survival (BRFS): T stage, LN RT dose, local RT dose, and ADT duration period. Any significantly different toxicity was not seen for each LN or local RT dose except for the incident rate of grade 2 or more acute urinary retention, which was significantly higher in the higher LN RT dose (60 Gy or more) group by the Chi-square test. RT dose escalation to metastatic LN in cN1 patients improves BRFS, RFS, and DMFS at 4 and 7 years, without increasing severe adverse events.

Sections du résumé

BACKGROUND
In this study, we aim to present the clinical outcomes of radiotherapy (RT) in clinical pelvic lymph node-positive prostate cancer (cN1) patients. We also analyze the prognostic factors with focus on RT dose escalation to metastatic lymph nodes (LN).
METHODS
We retrospectively analyzed the data from cN1 patients who were treated with definitive RT and androgen deprivation therapy (ADT) between June 2004 and February 2016. All patients received localized irradiation to the prostate region and whole pelvis irradiation. Some patients received intensity-modulated radiation therapy with RT dose escalation to metastatic LN. Univariate analyses using log-rank test were performed to find prognostic factors between patient subgroups.
RESULTS
Fifty-one consecutive patients were identified. The median follow-up period for all patients was 88 (range 20-157) months. Primary Gleason pattern and LN RT dose were statistically significant prognostic factors for relapse-free survival (RFS) and distant metastasis-free survival (DMFS). Especially, RT dose escalation (60 Gy or more) to metastatic LN significantly improved RFS and DMFS compared with standard dose RT (4-year RFS 90.6% vs 82.1%, 7-year RFS 90.6% vs 58.0%, P = .015; 4-year DMFS 90.6% vs 82.1%, 7-year DMFS 90.6% vs 62.8%, P = .023). The following factors were all statistically significant for biochemical relapse-free survival (BRFS): T stage, LN RT dose, local RT dose, and ADT duration period. Any significantly different toxicity was not seen for each LN or local RT dose except for the incident rate of grade 2 or more acute urinary retention, which was significantly higher in the higher LN RT dose (60 Gy or more) group by the Chi-square test.
CONCLUSIONS
RT dose escalation to metastatic LN in cN1 patients improves BRFS, RFS, and DMFS at 4 and 7 years, without increasing severe adverse events.

Identifiants

pubmed: 32750234
doi: 10.1002/cam4.2985
pmc: PMC7520291
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

6629-6637

Informations de copyright

© 2020 The Authors. Cancer Medicine published by John Wiley & Sons Ltd.

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Auteurs

Keisuke Tsuchida (K)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.
Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Japan.

Koji Inaba (K)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Tairo Kashihara (T)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Naoya Murakami (N)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Kae Okuma (K)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Kana Takahashi (K)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Hiroshi Igaki (H)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Yuko Nakayama (Y)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

Aiko Maejima (A)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Yasuo Shinoda (Y)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Yoshiyuki Matsui (Y)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Motokiyo Komiyama (M)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Hiroyuki Fujimoto (H)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Yoshinori Ito (Y)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.
Department of Radiation Oncology, Showa University School of Medicine, Tokyo, Japan.

Minako Sumi (M)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.
Radiation Oncology Department, Cancer Institute Hospital, Japanese Foundation for Cancer Research, Tokyo, Japan.

Takashi Nakano (T)

Department of Radiation Oncology, Gunma University Graduate School of Medicine, Maebashi, Japan.

Jun Itami (J)

Department of Radiation Oncology, National Cancer Center Hospital, Tokyo, Japan.

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Classifications MeSH