Orbitofrontal-striatal potentiation underlies cocaine-induced hyperactivity.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
10 08 2020
Historique:
received: 16 04 2020
accepted: 16 07 2020
entrez: 12 8 2020
pubmed: 12 8 2020
medline: 12 9 2020
Statut: epublish

Résumé

Psychomotor stimulants increase dopamine levels in the striatum and promote locomotion; however, their effects on striatal pathway function in vivo remain unclear. One model that has been proposed to account for these motor effects suggests that stimulants drive hyperactivity via activation and inhibition of direct and indirect pathway striatal neurons, respectively. Although this hypothesis is consistent with the cellular actions of dopamine receptors and received support from optogenetic and chemogenetic studies, it has been rarely tested with in vivo recordings. Here, we test this model and observe that cocaine increases the activity of both pathways in the striatum of awake mice. These changes are linked to a dopamine-dependent cocaine-induced strengthening of upstream orbitofrontal cortex (OFC) inputs to the dorsomedial striatum (DMS) in vivo. Finally, depressing OFC-DMS pathway with a high frequency stimulation protocol in awake mice over-powers the cocaine-induced potentiation of OFC-DMS pathway and attenuates the expression of locomotor sensitization, directly linking OFC-DMS potentiation to cocaine-induced hyperactivity.

Identifiants

pubmed: 32778725
doi: 10.1038/s41467-020-17763-8
pii: 10.1038/s41467-020-17763-8
pmc: PMC7417999
doi:

Substances chimiques

Central Nervous System Stimulants 0
Cocaine I5Y540LHVR
Dopamine VTD58H1Z2X

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, N.I.H., Intramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3996

Subventions

Organisme : NIDA NIH HHS
ID : R21 DA047127
Pays : United States

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Auteurs

Sebastiano Bariselli (S)

National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD, 20892, USA.
National Institute on Alcohol Abuse and Alcoholism (NIAAA), Laboratory for Integrative Neuroscience (LIN), Bethesda, MD, 20892-9412, USA.

Nanami L Miyazaki (NL)

National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD, 20892, USA.

Meaghan C Creed (MC)

Washington University Pain Center, St Louis, MO, 63110, USA.
Departments of Psychiatry, Anesthesiology, and Neuroscience, Washington University School of Medicine, St Louis, MO, 63110, USA.

Alexxai V Kravitz (AV)

National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD, 20892, USA. alexxai@email.wustl.edu.
Departments of Psychiatry, Anesthesiology, and Neuroscience, Washington University School of Medicine, St Louis, MO, 63110, USA. alexxai@email.wustl.edu.

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