Vascular Remodeling in Moyamoya Angiopathy: From Peripheral Blood Mononuclear Cells to Endothelial Cells.
Adult
Biomarkers
/ blood
Cell Count
Child
Cytokines
/ metabolism
Endothelial Cells
/ pathology
Female
Gene Expression Regulation
Human Umbilical Vein Endothelial Cells
/ metabolism
Humans
Leukocytes, Mononuclear
/ pathology
Male
Moyamoya Disease
/ blood
Neovascularization, Physiologic
Paracrine Communication
RNA, Messenger
/ genetics
Vascular Remodeling
/ genetics
Moyamoya angiopathy
RNF213
endothelial progenitor cells
neovascularization
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
11 Aug 2020
11 Aug 2020
Historique:
received:
02
07
2020
revised:
30
07
2020
accepted:
06
08
2020
entrez:
16
8
2020
pubmed:
17
8
2020
medline:
18
2
2021
Statut:
epublish
Résumé
The pathophysiological mechanisms of Moyamoya angiopathy (MA), which is a rare cerebrovascular condition characterized by recurrent ischemic/hemorrhagic strokes, are still largely unknown. An imbalance of vasculogenic/angiogenic mechanisms has been proposed as one possible disease aspect. Circulating endothelial progenitor cells (cEPCs) have been hypothesized to contribute to vascular remodeling of MA, but it remains unclear whether they might be considered a disease effect or have a role in disease pathogenesis. The aim of the present study was to provide a morphological, phenotypical, and functional characterization of the cEPCs from MA patients to uncover their role in the disease pathophysiology. cEPCs were identified from whole blood as CD45
Identifiants
pubmed: 32796702
pii: ijms21165763
doi: 10.3390/ijms21165763
pmc: PMC7460840
pii:
doi:
Substances chimiques
Biomarkers
0
Cytokines
0
RNA, Messenger
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
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