Viral adaption of staphylococcal phage: A genome-based analysis of the selective preference based on codon usage Bias.


Journal

Genomics
ISSN: 1089-8646
Titre abrégé: Genomics
Pays: United States
ID NLM: 8800135

Informations de publication

Date de publication:
11 2020
Historique:
received: 04 03 2020
revised: 19 07 2020
accepted: 11 08 2020
pubmed: 21 8 2020
medline: 14 9 2021
entrez: 21 8 2020
Statut: ppublish

Résumé

Given the high therapeutic value of the staphylococcal phage, the genome co-evolution of the phage and the host has gained great attention. Though the genome-wide AT richness in staphylococcal phages has been well-studied with nucleotide usage bias, here we proved that host factor, lifestyle and taxonomy are also important factors in understanding the phage nucleotide usages bias using information entropy formula. Such correlation is especially prominent when it comes to the synonymous codon usages of staphylococcal phages, despite the overall scattered codon usage pattern represented by principal component analysis. This strong relationship is explained by nucleotide skew which testified that the usage biases of nucleotide at different codon positions are acting on synonymous codons. Therefore, our study reveals a hidden relationship of genome evolution with host limitation and phagic phenotype, providing new insight into phage genome evolution at genetic level.

Identifiants

pubmed: 32818632
pii: S0888-7543(20)30216-0
doi: 10.1016/j.ygeno.2020.08.012
pii:
doi:

Substances chimiques

Nucleotides 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

4657-4665

Informations de copyright

Copyright © 2020 Elsevier Inc. All rights reserved.

Auteurs

Zhiyi Ge (Z)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Xuerui Li (X)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Xiaoan Cao (X)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Rui Wang (R)

Viterbi School of Engineering, University of Southern California, Los Angeles, CA 90089, United States of America.

Wen Hu (W)

Gansu Police Vocational College, Lanzhou 730046, Gansu, PR China.

Ling Gen (L)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Shengyi Han (S)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China; The College of Veterinary Medicine, Gansu Agricultural University, Lanzhou 730070, Gansu Province, PR China.

Youjun Shang (Y)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Yongsheng Liu (Y)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China.

Jian-Hua Zhou (JH)

State Key Laboratory of Veterinary Etiological Biology, Lanzhou Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Lanzhou 730046, Gansu, PR China. Electronic address: zhoujianhuazjh@163.com.

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