Sevoflurane depresses neurons in the medial parabrachial nucleus by potentiating postsynaptic GABA


Journal

Neuropharmacology
ISSN: 1873-7064
Titre abrégé: Neuropharmacology
Pays: England
ID NLM: 0236217

Informations de publication

Date de publication:
15 12 2020
Historique:
received: 12 01 2020
revised: 23 06 2020
accepted: 20 07 2020
pubmed: 16 9 2020
medline: 24 9 2021
entrez: 15 9 2020
Statut: ppublish

Résumé

Despite persistent clinical use for over 170 years, the neuronal mechanisms by which general anesthetics produce hypnosis remain unclear. Previous studies suggest that anesthetics exert hypnotic effects by acting on endogenous arousal circuits. Recently, it has been shown that the medial parabrachial nucleus (MPB) is a novel wake-promoting component in the dorsolateral pons. However, it is not known whether and how the MPB contributes to anesthetic-induced hypnosis. Here, we investigated the action of sevoflurane, a widely used volatile anesthetic agent that best represents the drug class of halogenated ethers, on MPB neurons in mice. Using in vivo fiber photometry, we found that the population activities of MPB neurons were inhibited during sevoflurane-induced loss of consciousness. Using in vitro whole-cell patch-clamp recordings, we revealed that sevoflurane suppressed the firing rate of MPB neurons in concentration-dependent and reversible manners. At a concentration equal to MAC of hypnosis, sevoflurane potentiated synaptic GABA

Identifiants

pubmed: 32931816
pii: S0028-3908(20)30317-8
doi: 10.1016/j.neuropharm.2020.108249
pii:
doi:

Substances chimiques

Anesthetics, Inhalation 0
GABA Antagonists 0
Potassium Channels 0
Receptors, GABA-A 0
Picrotoxin 124-87-8
Sevoflurane 38LVP0K73A

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

108249

Informations de copyright

Copyright © 2020. Published by Elsevier Ltd.

Auteurs

Wei Xu (W)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China.

Lu Wang (L)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China.

Xiang-Shan Yuan (XS)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China.

Tian-Xiao Wang (TX)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China.

Wen-Xian Li (WX)

Department of Anesthesiology, The Eye, Ear, Nose and Throat Hospital of Fudan University, Shanghai Medical College of Fudan University, Shanghai, China.

Wei-Min Qu (WM)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China.

Zong-Yuan Hong (ZY)

Laboratory of Quantitative Pharmacology, Department of Pharmacology, Wannan Medical College, Wuhu, China.

Zhi-Li Huang (ZL)

Department of Pharmacology, School of Basic Medical Sciences; State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, and Institutes of Brain Science, Shanghai Medical College of Fudan University, Shanghai, China. Electronic address: huangzl@fudan.edu.cn.

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Classifications MeSH