Interferon Regulatory Factor 7 Attenuates Chronic Gammaherpesvirus Infection.


Journal

Journal of virology
ISSN: 1098-5514
Titre abrégé: J Virol
Pays: United States
ID NLM: 0113724

Informations de publication

Date de publication:
23 11 2020
Historique:
received: 29 07 2020
accepted: 15 09 2020
pubmed: 25 9 2020
medline: 26 1 2021
entrez: 24 9 2020
Statut: epublish

Résumé

Gammaherpesviruses are ubiquitous pathogens that establish lifelong infections and are associated with a variety of malignancies, including lymphomas. Interferon regulatory factor 7 (IRF-7) is an innate immune transcription factor that restricts acute replication of diverse viruses, including murine gammaherpesvirus 68 (MHV68). Importantly, very little is known about the role of IRF-7 during chronic virus infections. In this study, we demonstrate that IRF-7 attenuates chronic infection by restricting establishment of gammaherpesvirus latency in the peritoneal cavity and, to a lesser extent, viral reactivation in the spleen. Despite the classical role of IRF-7 as a stimulator of type I interferon (IFN) transcription, there were no global effects on the expression of IFN-induced genes (ISGs) in the absence of IRF-7, with only a few ISGs showing attenuated expression in IRF-7-deficient peritoneal cells. Further, IRF-7 expression was dispensable for the induction of a virus-specific CD8 T cell response. In contrast, IRF-7 expression restricted latent gammaherpesvirus infection in the peritoneal cavity under conditions where the viral latent reservoir is predominantly hosted by peritoneal B cells. This report is the first demonstration of the antiviral role of IRF-7 during the chronic stage of gammaherpesvirus infection.

Identifiants

pubmed: 32967960
pii: JVI.01554-20
doi: 10.1128/JVI.01554-20
pmc: PMC7925175
pii:
doi:

Substances chimiques

Interferon Regulatory Factor-7 0
Interferon Type I 0
Irf7 protein, mouse 0
ADAR1 protein, mouse EC 3.5.4.4
Adenosine Deaminase EC 3.5.4.4

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NCI NIH HHS
ID : R01 CA183593
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA203923
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM080202
Pays : United States
Organisme : NCI NIH HHS
ID : F31 CA243364
Pays : United States
Organisme : NCI NIH HHS
ID : F30 CA247000
Pays : United States

Informations de copyright

Copyright © 2020 American Society for Microbiology.

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Auteurs

K E Johnson (KE)

Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.

C A Aurubin (CA)

Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.

C N Jondle (CN)

Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.

P T Lange (PT)

Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.

V L Tarakanova (VL)

Department of Microbiology and Immunology, Medical College of Wisconsin, Milwaukee, Wisconsin, USA vera@mcw.edu.
Cancer Center, Medical College of Wisconsin, Milwaukee, Wisconsin, USA.

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