Anxiety in Parkinson's disease is associated with changes in the brain fear circuit.


Journal

Parkinsonism & related disorders
ISSN: 1873-5126
Titre abrégé: Parkinsonism Relat Disord
Pays: England
ID NLM: 9513583

Informations de publication

Date de publication:
11 2020
Historique:
received: 19 03 2020
revised: 23 08 2020
accepted: 14 09 2020
pubmed: 27 9 2020
medline: 25 2 2023
entrez: 26 9 2020
Statut: ppublish

Résumé

Anxiety is frequent in Parkinson's disease (PD) and has a negative impact on disease symptoms and quality of life. The underlying mechanisms remain largely unknown. The aim of this study was to identify anatomical and functional changes associated to PD-related anxiety by comparing the volume, shape and texture of the amygdala, the cortical thickness as well as the functional connectivity (FC) of the fear circuit in patients with and without clinically relevant anxiety. Non-demented PD patients were recruited, and anxiety was quantified using the Parkinson Anxiety Scale. Structural MRI was used to compare cortical thickness and amygdala structure and resting-state functional MRI to compare FC patterns of the amygdala and resting-state functional networks in both groups. We included 118 patients: 34 with (A+) and 84 without (A-) clinically relevant anxiety. Clusters of cortical thinning were identified in the bilateral fronto-cingulate and left parietal cortices of the A+ group. The texture and the shape of the left amygdala was different in the A+ group but the overall volume did not differ between groups. FC between the amygdala and the whole brain regions did not differ between groups. The internetwork resting-state FC was higher between the "fear circuit" and salience network in the A+ group. Anxiety in PD induces structural modifications of the left amygdala, atrophy of the bilateral fronto-cingulate and the left parietal cortices, and a higher internetwork resting-state FC between the fear circuit and the salience network.

Sections du résumé

BACKGROUND
Anxiety is frequent in Parkinson's disease (PD) and has a negative impact on disease symptoms and quality of life. The underlying mechanisms remain largely unknown. The aim of this study was to identify anatomical and functional changes associated to PD-related anxiety by comparing the volume, shape and texture of the amygdala, the cortical thickness as well as the functional connectivity (FC) of the fear circuit in patients with and without clinically relevant anxiety.
METHODS
Non-demented PD patients were recruited, and anxiety was quantified using the Parkinson Anxiety Scale. Structural MRI was used to compare cortical thickness and amygdala structure and resting-state functional MRI to compare FC patterns of the amygdala and resting-state functional networks in both groups.
RESULTS
We included 118 patients: 34 with (A+) and 84 without (A-) clinically relevant anxiety. Clusters of cortical thinning were identified in the bilateral fronto-cingulate and left parietal cortices of the A+ group. The texture and the shape of the left amygdala was different in the A+ group but the overall volume did not differ between groups. FC between the amygdala and the whole brain regions did not differ between groups. The internetwork resting-state FC was higher between the "fear circuit" and salience network in the A+ group.
CONCLUSION
Anxiety in PD induces structural modifications of the left amygdala, atrophy of the bilateral fronto-cingulate and the left parietal cortices, and a higher internetwork resting-state FC between the fear circuit and the salience network.

Identifiants

pubmed: 32979785
pii: S1353-8020(20)30734-3
doi: 10.1016/j.parkreldis.2020.09.020
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT01792843']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

89-97

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Guillaume Carey (G)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France. Electronic address: guillaume.carey@chru-lille.fr.

Renaud Lopes (R)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France; Department of Neuroradiology, Lille University Medical Centre, Lille, France.

Romain Viard (R)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France; Department of Neuroradiology, Lille University Medical Centre, Lille, France.

Nacim Betrouni (N)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France.

Gregory Kuchcinski (G)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France; Department of Neuroradiology, Lille University Medical Centre, Lille, France.

Quentin Devignes (Q)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France.

Luc Defebvre (L)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France; Neurology and Movement Disorders Department, Lille University Medical Centre, Lille, France.

Albert F G Leentjens (AFG)

Department of Psychiatry, Maastricht University Medical Centre, Maastricht, the Netherlands.

Kathy Dujardin (K)

Univ. Lille, Inserm, CHU Lille, Lille Neurosciences and Cognition, Lille, France; Neurology and Movement Disorders Department, Lille University Medical Centre, Lille, France.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH