Neutrophilic inflammation in the respiratory mucosa predisposes to RSV infection.


Journal

Science (New York, N.Y.)
ISSN: 1095-9203
Titre abrégé: Science
Pays: United States
ID NLM: 0404511

Informations de publication

Date de publication:
09 10 2020
Historique:
received: 17 01 2020
revised: 30 06 2020
accepted: 19 08 2020
entrez: 9 10 2020
pubmed: 10 10 2020
medline: 29 10 2020
Statut: ppublish

Résumé

The variable outcome of viral exposure is only partially explained by known factors. We administered respiratory syncytial virus (RSV) to 58 volunteers, of whom 57% became infected. Mucosal neutrophil activation before exposure was highly predictive of symptomatic RSV disease. This was associated with a rapid, presymptomatic decline in mucosal interleukin-17A (IL-17A) and other mediators. Conversely, those who resisted infection showed presymptomatic activation of IL-17- and tumor necrosis factor-related pathways. Vulnerability to infection was not associated with baseline microbiome but was reproduced in mice by preinfection chemokine-driven airway recruitment of neutrophils, which caused enhanced disease mediated by pulmonary CD8

Identifiants

pubmed: 33033192
pii: 370/6513/eaba9301
doi: 10.1126/science.aba9301
pmc: PMC7613218
mid: EMS145175
pii:
doi:

Substances chimiques

Chemokine CXCL1 0
Interleukin-17 0
Tumor Necrosis Factor-alpha 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Medical Research Council
ID : MR/T50256X/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : G0902266
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 087805
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 090382
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/R005982/1
Pays : United Kingdom
Organisme : Medical Research Council
ID : MR/R502121/1
Pays : United Kingdom
Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 109058
Pays : United Kingdom

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020, American Association for the Advancement of Science.

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Auteurs

Maximillian S Habibi (MS)

National Heart and Lung Institute, Imperial College London, London, UK.

Ryan S Thwaites (RS)

National Heart and Lung Institute, Imperial College London, London, UK.

Meiping Chang (M)

Merck & Co., Inc., Kenilworth, NJ, USA.

Agnieszka Jozwik (A)

National Heart and Lung Institute, Imperial College London, London, UK.

Allan Paras (A)

National Heart and Lung Institute, Imperial College London, London, UK.

Freja Kirsebom (F)

National Heart and Lung Institute, Imperial College London, London, UK.

Augusto Varese (A)

National Heart and Lung Institute, Imperial College London, London, UK.

Amber Owen (A)

National Heart and Lung Institute, Imperial College London, London, UK.

Leah Cuthbertson (L)

National Heart and Lung Institute, Imperial College London, London, UK.

Phillip James (P)

National Heart and Lung Institute, Imperial College London, London, UK.

Tanushree Tunstall (T)

National Heart and Lung Institute, Imperial College London, London, UK.

David Nickle (D)

Genetics & Pharmacogenomics, Department of Translational Medicine, Merck & Co., Inc., Boston, MA, USA.

Trevor T Hansel (TT)

National Heart and Lung Institute, Imperial College London, London, UK.

Miriam F Moffatt (MF)

National Heart and Lung Institute, Imperial College London, London, UK.

Cecilia Johansson (C)

National Heart and Lung Institute, Imperial College London, London, UK. p.openshaw@imperial.ac.uk c.chiu@imperial.ac.uk c.johansson@imperial.ac.uk.

Christopher Chiu (C)

Department of Infectious Disease, Imperial College London, London, UK. p.openshaw@imperial.ac.uk c.chiu@imperial.ac.uk c.johansson@imperial.ac.uk.

Peter J M Openshaw (PJM)

National Heart and Lung Institute, Imperial College London, London, UK. p.openshaw@imperial.ac.uk c.chiu@imperial.ac.uk c.johansson@imperial.ac.uk.

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Classifications MeSH