Astrogliosis in an Experimental Model of Hypovitaminosis B12: A Cellular Basis of Neurological Disorders due to Cobalamin Deficiency.
astrocytes
astrogliosis
cobalamin
hypovitaminosis B12
neurological disorders
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
09 10 2020
09 10 2020
Historique:
received:
16
09
2020
revised:
06
10
2020
accepted:
07
10
2020
entrez:
14
10
2020
pubmed:
15
10
2020
medline:
3
7
2021
Statut:
epublish
Résumé
Cobalamin deficiency affects human physiology with sequelae ranging from mild fatigue to severe neuropsychiatric abnormalities. The cellular and molecular aspects of the nervous system disorders associated with hypovitaminosis B12 remain largely unknown. Growing evidence indicates that astrogliosis is an underlying component of a wide range of neuropathologies. Previously, we developed an in vitro model of cobalamin deficiency in normal human astrocytes (NHA) by culturing the cells with
Identifiants
pubmed: 33050187
pii: cells9102261
doi: 10.3390/cells9102261
pmc: PMC7600008
pii:
doi:
Substances chimiques
Biomarkers
0
Glial Fibrillary Acidic Protein
0
Vimentin
0
Vitamin B 12
P6YC3EG204
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
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