Serum potassium dynamics during acute heart failure hospitalization.


Journal

Clinical research in cardiology : official journal of the German Cardiac Society
ISSN: 1861-0692
Titre abrégé: Clin Res Cardiol
Pays: Germany
ID NLM: 101264123

Informations de publication

Date de publication:
Apr 2022
Historique:
received: 01 08 2020
accepted: 30 09 2020
pubmed: 19 10 2020
medline: 5 4 2022
entrez: 18 10 2020
Statut: ppublish

Résumé

Available information about prognostic implications of potassium levels alteration in the setting of acute heart failure (AHF) is scarce. We aim to describe the prevalence of dyskalemia (hypo or hyperkalemia), its dynamic changes during AHF-hospitalization, and its long-term clinical impact after hospitalization. We analyzed 1779 patients hospitalized with AHF who were included in the REDINSCOR II registry. Patients were classified in three groups, according to potassium levels both on admission and discharge: hypokalemia (potassium < 3.5 mEq/L), normokalemia (potassium = 3.5-5.0 mEq/L and, hyperkalemia (potassium > 5 mEq/L). The prevalence of hypokalemia and hyperkalemia on admission was 8.2 and 4.6%, respectively, and 6.4 and 2.7% at discharge. Hyperkalemia on admission was associated with higher in-hospital mortality (OR = 2.32 [95% CI: 1.04-5.21] p = 0.045). Among patients with hypokalemia on admission, 79% had normalized potassium levels at discharge. In the case of patients with hyperkalemia on admission, 89% normalized kalemia before discharge. In multivariate Cox regression, dyskalemia was associated with higher 12-month mortality, (HR = 1.48 [95% CI, 1.12-1.96], p = 0.005). Among all patterns of dyskalemia persistent hypokalemia (HR = 3.17 [95% CI: 1.71-5.88]; p < 0.001), and transient hyperkalemia (HR = 1.75 [95% CI: 1.07-2.86]; p = 0.023) were related to reduced 12-month survival. Potassium levels alterations are frequent and show a dynamic behavior during AHF admission. Hyperkalemia on admission is an independent predictor of higher in-hospital mortality. Furthermore, persistent hypokalemia and transient hyperkalemia on admission are independent predictors of 12-month mortality.

Sections du résumé

BACKGROUND BACKGROUND
Available information about prognostic implications of potassium levels alteration in the setting of acute heart failure (AHF) is scarce.
OBJECTIVES OBJECTIVE
We aim to describe the prevalence of dyskalemia (hypo or hyperkalemia), its dynamic changes during AHF-hospitalization, and its long-term clinical impact after hospitalization.
METHODS METHODS
We analyzed 1779 patients hospitalized with AHF who were included in the REDINSCOR II registry. Patients were classified in three groups, according to potassium levels both on admission and discharge: hypokalemia (potassium < 3.5 mEq/L), normokalemia (potassium = 3.5-5.0 mEq/L and, hyperkalemia (potassium > 5 mEq/L).
RESULTS RESULTS
The prevalence of hypokalemia and hyperkalemia on admission was 8.2 and 4.6%, respectively, and 6.4 and 2.7% at discharge. Hyperkalemia on admission was associated with higher in-hospital mortality (OR = 2.32 [95% CI: 1.04-5.21] p = 0.045). Among patients with hypokalemia on admission, 79% had normalized potassium levels at discharge. In the case of patients with hyperkalemia on admission, 89% normalized kalemia before discharge. In multivariate Cox regression, dyskalemia was associated with higher 12-month mortality, (HR = 1.48 [95% CI, 1.12-1.96], p = 0.005). Among all patterns of dyskalemia persistent hypokalemia (HR = 3.17 [95% CI: 1.71-5.88]; p < 0.001), and transient hyperkalemia (HR = 1.75 [95% CI: 1.07-2.86]; p = 0.023) were related to reduced 12-month survival.
CONCLUSIONS CONCLUSIONS
Potassium levels alterations are frequent and show a dynamic behavior during AHF admission. Hyperkalemia on admission is an independent predictor of higher in-hospital mortality. Furthermore, persistent hypokalemia and transient hyperkalemia on admission are independent predictors of 12-month mortality.

Identifiants

pubmed: 33070219
doi: 10.1007/s00392-020-01753-3
pii: 10.1007/s00392-020-01753-3
doi:

Substances chimiques

Potassium RWP5GA015D

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

368-379

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2020. Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Pedro Caravaca Perez (P)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario 12 de Octubre, Instituto de Investigación Sanitaria Hospital 12 Octubre (imas12), Avenida de Andalucía s/n, 28041, Madrid, Spain.

José R González-Juanatey (JR)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Complejo Hospitalario Universitario de Santiago de Compostela, Facultad de Medicina, Universidad de Santiago, Santiago de Compostela, Spain.

Jorge Nuche (J)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Centro Nacional de Investigaciones Cardiovasculares, Madrid, Spain.

Laura Morán Fernández (L)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario 12 de Octubre, Instituto de Investigación Sanitaria Hospital 12 Octubre (imas12), Avenida de Andalucía s/n, 28041, Madrid, Spain.

David Lora Pablos (D)

Instituto de Investigación Sanitaria Hospital 12 Octubre (imas12), Universidad Complutense de Madrid, CIBERESP, Madrid, Spain.

Jesús Alvarez-García (J)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario Santa Creu i Sant Pau, Universidad de Barcelona, Barcelona, Spain.

Ramón Bascompte Claret (R)

Hospital Universitari Arnau de Vilanova, IBRLLEIDA, Lleida, Spain.

Manuel Martínez Selles (M)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario Gregorio Marañón, Instituto de Investigación Sanitaria IiGM, Universidad Europea, Madrid, Spain.

Rafael Vázquez García (R)

UGC de Cardiología, Hospital Universitario Puerta del Mar, Cádiz, Spain.

Luis Martínez Dolz (L)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario Y Politécnico La Fe, IIS La Fe, Valencia, Spain.

Marta Cobo-Marcos (M)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario Puerta de Hierro, Madrid, Spain.

Domingo Pascual Figal (D)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Hospital Universitario Virgen de La Arrixaca, Universidad de Murcia, Murcia, Spain.

Maria G Crespo-Leiro (MG)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Department of Cardiology, Complexo Hospitalario Universitario A Coruña (CHUAC), Instituto de Investigación Biomedica A Coruña (INIBIC), Universidade da Coruña (UDC), A Coruña, Spain.

Julio Nuñez Villota (J)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain.
Servicio de Cardiología, Hospital Clinico Universitario de Valencia, INCLIVA, Universitat de Valencia, Valencia, Spain.

Juan Cinca Cuscullola (J)

Centro Nacional de Investigaciones Cardiovasculares, Madrid, Spain.

Juan F Delgado (JF)

CIBER de Enfermedades Cardiovasculares (CIBERCV), Madrid, Spain. juan.delgado@salud.madrid.org.
Department of Cardiology, Hospital Universitario 12 de Octubre, Instituto de Investigación Sanitaria Hospital 12 Octubre (imas12), Avenida de Andalucía s/n, 28041, Madrid, Spain. juan.delgado@salud.madrid.org.
Facultad de Medicina, Universidad Complutense de Madrid (UCM), Madrid, Spain. juan.delgado@salud.madrid.org.

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