Two frequent loss-of-function mutations in Aurora Kinase C gene in Algerian infertile men with macrozoospermia.

AURKC mutations ICSI outcomes macrozoospermia male infertility nonsense mutation

Journal

Andrologia
ISSN: 1439-0272
Titre abrégé: Andrologia
Pays: Germany
ID NLM: 0423506

Informations de publication

Date de publication:
Dec 2020
Historique:
received: 17 06 2020
revised: 06 09 2020
accepted: 10 09 2020
pubmed: 30 10 2020
medline: 22 6 2021
entrez: 29 10 2020
Statut: ppublish

Résumé

Macrozoospermia is associated with severe male infertility. To date, the only gene implicated in this phenotype is the Aurora Kinase C gene. We report in this work the genetic screening of AURKC mutations in 34 patients with macrozoospermia among 3,536 Algerian infertile men. Nineteen patients (56%) were homozygotes for the c.144delC mutation, eight (23.52%) homozygotes for the c.744C>G (p.Y248*) mutation and two (5.88%) compound heterozygotes. No AURKC mutation was identified in five patients (14.7%). Interestingly and although it is generally accepted that nearly all positive mutated AURKC patients have close to 100% large-head spermatozoa, our results showed that 11 patients with AURKC mutations (32.35%) had large-headed spermatozoa lower than 70% (7 with c.144delC and 4 with p.Y248*), and no mutation was found in 2 patients who had >70% of macrocephalic spermatozoa. Twenty ICSI attempts were performed before genetic screening resulting in 39 embryos but no pregnancy was obtained. The sequencing of AURKC exons 3 and 6 is appropriate as a first-line genetic exploration in these patients to avoid unsuccessful ICSI attempts. A percentage of large head spermatozoa beyond 25% and a percentage of multiflagellar spermatozoa beyond 10% are predictive of a positive mutation diagnosis.

Identifiants

pubmed: 33118205
doi: 10.1111/and.13868
doi:

Substances chimiques

AURKC protein, human EC 2.7.11.1
Aurora Kinase C EC 2.7.11.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13868

Informations de copyright

© 2020 Wiley-VCH GmbH.

Références

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Auteurs

Loubna Hamza (L)

Faculté des Sciences Biologiques, Université de Science et de Technologie Houari Boumediane (USTHB), Bab Ezzouar, Algeria.
Centre d'Assistance Médicale à la Procréation Tiziri, Alger, Algeria.

Natacha Gaitch (N)

Assistance Publique - Hôpitaux de Paris, Site Cochin, Laboratoire de Génétique et Biologie Moléculaires, Groupe Universitaire Paris Centre, Paris, France.

Amira Sallem (A)

Assistance Publique - Hôpitaux de Paris, Site Cochin, Service d'Histologie, Embryologie, Biologie de la Reproduction - CECOS, Groupe Universitaire Paris Centre, Paris, France.
Inserm, U1016, Institut Cochin, Paris, France.
Université Paris Descartes, Sorbonne Paris Cité, Paris, France.
Laboratoire d'Histologie-Embryologie et de Cytogénétique (LR18ES40), Faculté de Médecine de Monastir, Université de Monastir, Monastir, Tunisie.

Nadjia Boucekkine (N)

Centre d'Assistance Médicale à la Procréation Tiziri, Alger, Algeria.

Emmanuelle Girodon (E)

Assistance Publique - Hôpitaux de Paris, Site Cochin, Laboratoire de Génétique et Biologie Moléculaires, Groupe Universitaire Paris Centre, Paris, France.

Amina Oumeziane (A)

Centre d'Assistance Médicale à la Procréation Tiziri, Alger, Algeria.

Nabila Attal (N)

Institut Pasteur d'Algérie ; service d'Immunologie, Dely Ibrahim, Alger, Algeria.

Jean Philippe Wolf (JP)

Assistance Publique - Hôpitaux de Paris, Site Cochin, Service d'Histologie, Embryologie, Biologie de la Reproduction - CECOS, Groupe Universitaire Paris Centre, Paris, France.
Inserm, U1016, Institut Cochin, Paris, France.
Université Paris Descartes, Sorbonne Paris Cité, Paris, France.
CNRS, UMR8104, Paris, France.

Thierry Bienvenu (T)

Assistance Publique - Hôpitaux de Paris, Site Cochin, Laboratoire de Génétique et Biologie Moléculaires, Groupe Universitaire Paris Centre, Paris, France.
Inserm, U1016, Institut Cochin, Paris, France.
Université Paris Descartes, Sorbonne Paris Cité, Paris, France.
CNRS, UMR8104, Paris, France.

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