Negative Food Effect of Danirixin: Use of PBPK Modelling to Explore the Effect of Formulation and Meal Type on Clinical PK.


Journal

Pharmaceutical research
ISSN: 1573-904X
Titre abrégé: Pharm Res
Pays: United States
ID NLM: 8406521

Informations de publication

Date de publication:
29 Oct 2020
Historique:
received: 28 02 2020
accepted: 07 10 2020
entrez: 30 10 2020
pubmed: 31 10 2020
medline: 7 8 2021
Statut: epublish

Résumé

To use physiologically-based pharmacokinetic (PBPK) modelling to explore the food effect of different DNX hydrobromide (HBr) hemihydrate salt tablet formulations using biorelevant dissolution. Compendial dissolution using a paddle method and TIM-1 biorelevant dissolution were performed and incorporated into a previously reported PBPK model. A two-part clinical study evaluated tablet formulations in the fasted/fed (high fat) state (Part A), and the impact of food (fasted/normal/high fat) and Proton Pump Inhibitor (PPI) co-administration for a selected formulation; as well as a formulation containing DNX HBr in the monohydrate state (Part B). TIM-1 data showed that the fed state bioaccessibility of DNX was significantly decreased compared to the fasted state with no significant differences between formulations. Dosed with normal/high fat food the selected formulation showed comparable exposure and a modest increase in DNX systemic PK was observed with PPI dependent on meal type. Under fed conditions DNX systemic exposure was comparable for the monohydrate and hemihydrate formulations. The integration of biorelevant TIM-1 data into the PBPK model led to the successful simulation of a DNX negative food effect. Interactions between DNX and food components are the likely the source of the negative food effect via micellar entrapment, ion pairing and/or meal induced viscosity changes.

Identifiants

pubmed: 33123802
doi: 10.1007/s11095-020-02948-z
pii: 10.1007/s11095-020-02948-z
doi:

Substances chimiques

Piperidines 0
Proton Pump Inhibitors 0
Sulfones 0
Tablets 0
danirixin R318PGH5VP

Types de publication

Clinical Trial, Phase I Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

233

Références

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Auteurs

Richard S Lloyd (RS)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

Martin I Hingle (MI)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

Jackie C Bloomer (JC)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

Stephen J Charles (SJ)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

James M Butler (JM)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

Alan Paul (A)

Medicinal Science and Technology, GlaxoSmithKline R&D, Park Road, Hertfordshire, UK.

Xiaofeng Zhu (X)

Medicinal Science and Technology, GlaxoSmithKline R&D, Collegeville, Pennsylvania, USA.

Bruce Miller (B)

Respiratory Therapy Area Unit, GlaxoSmithKline R&D, King of Prussia, Pennsylvania, USA.

Donald D'Amico (D)

Respiratory Therapy Area Unit, GlaxoSmithKline R&D, King of Prussia, Pennsylvania, USA.

Alison Donald (A)

Respiratory Therapy Area Unit, GlaxoSmithKline R&D, King of Prussia, Pennsylvania, USA.

Ruth Tal-Singer (R)

Medical Innovation, GlaxoSmithKline R&D, Collegeville, Pennsylvania, USA.

Claire Ambery (C)

Clinical Pharmacology and Modelling Simulation, GlaxoSmithKline R&D, 980 Great West Road, TW8 9GS, Brentford, UK. Claire.l.ambery@gsk.com.

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