Atg43 tethers isolation membranes to mitochondria to promote starvation-induced mitophagy in fission yeast.


Journal

eLife
ISSN: 2050-084X
Titre abrégé: Elife
Pays: England
ID NLM: 101579614

Informations de publication

Date de publication:
03 11 2020
Historique:
received: 20 07 2020
accepted: 09 10 2020
entrez: 3 11 2020
pubmed: 4 11 2020
medline: 15 5 2021
Statut: epublish

Résumé

Degradation of mitochondria through mitophagy contributes to the maintenance of mitochondrial function. In this study, we identified that Atg43, a mitochondrial outer membrane protein, serves as a mitophagy receptor in the model organism

Identifiants

pubmed: 33138913
doi: 10.7554/eLife.61245
pii: 61245
pmc: PMC7609059
doi:
pii:

Substances chimiques

Atg43 protein, S pombe 0
Autophagy-Related Protein 8 Family 0
Autophagy-Related Proteins 0
Mitochondrial Proteins 0
Schizosaccharomyces pombe Proteins 0
autophagy associated protein Atg8, S pombe 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Japan Society for the Promotion of Science
ID : 17K07330
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 20K06552
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 18H04858
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 19H05712
Pays : International
Organisme : Japan Society for the Promotion of Science
ID : 19K22419
Pays : International
Organisme : Japan Agency for Medical Research and Development
ID : 20gm6110013h0003
Pays : International

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2020, Fukuda et al.

Déclaration de conflit d'intérêts

TF, YE, TS, KF, SY, KI, DK, YY, TK No competing interests declared

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Auteurs

Tomoyuki Fukuda (T)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Yuki Ebi (Y)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Tetsu Saigusa (T)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Kentaro Furukawa (K)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Shun-Ichi Yamashita (SI)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Keiichi Inoue (K)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Daiki Kobayashi (D)

Omics Unit, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Yutaka Yoshida (Y)

Department of Structural Pathology, Kidney Research Center, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Tomotake Kanki (T)

Department of Cellular Physiology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

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