Overexpression of TGF-β1 induces renal fibrosis and accelerates the decline in kidney function in polycystic kidney disease.


Journal

American journal of physiology. Renal physiology
ISSN: 1522-1466
Titre abrégé: Am J Physiol Renal Physiol
Pays: United States
ID NLM: 100901990

Informations de publication

Date de publication:
01 12 2020
Historique:
pubmed: 10 11 2020
medline: 1 1 2021
entrez: 9 11 2020
Statut: ppublish

Résumé

Autosomal dominant polycystic kidney disease (ADPKD) is characterized by the presence of numerous fluid-filled cysts, extensive fibrosis, and the progressive decline in kidney function. Transforming growth factor-β1 (TGF-β1), an important mediator for renal fibrosis and chronic kidney disease, is overexpressed by cystic cells compared with normal kidney cells; however, its role in PKD pathogenesis remains undefined. To investigate the effect of TGF-β1 on cyst growth, fibrosis, and disease progression, we overexpressed active TGF-β1 specifically in collecting ducts (CDs) of phenotypic normal (

Identifiants

pubmed: 33166182
doi: 10.1152/ajprenal.00366.2020
pmc: PMC7792699
doi:

Substances chimiques

Pkhd1 protein, mouse 0
Receptors, Cell Surface 0
Transforming Growth Factor beta1 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

F1135-F1148

Subventions

Organisme : NIDDK NIH HHS
ID : P30 DK106912
Pays : United States
Organisme : NIDDK NIH HHS
ID : P30 DK079328
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK081579
Pays : United States
Organisme : NIDDK NIH HHS
ID : U54 DK126126
Pays : United States
Organisme : HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
ID : P30DK106912
Pays : International

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Auteurs

Yan Zhang (Y)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.

Yuqiao Dai (Y)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.

Archana Raman (A)

Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.
Department of Molecular and Integrative Physiology, University of Kansas Medical Center, Kansas City, Kansas.

Emily Daniel (E)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.

July Metcalf (J)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.

Gail Reif (G)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.

Fernando Pierucci-Alves (F)

Department of Anatomy and Physiology, Kansas State University, Manhattan, Kansas.

Darren P Wallace (DP)

Department of Internal Medicine, University of Kansas Medical Center, Kansas City, Kansas.
Jared Grantham Kidney Institute, University of Kansas Medical Center, Kansas City, Kansas.
Department of Molecular and Integrative Physiology, University of Kansas Medical Center, Kansas City, Kansas.

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Classifications MeSH