INSAID Variant Classification and Eurofever Criteria Guide Optimal Treatment Strategy in Patients with TRAPS: Data from the Eurofever Registry.


Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
02 2021
Historique:
received: 03 06 2020
revised: 23 09 2020
accepted: 26 10 2020
pubmed: 13 11 2020
medline: 22 5 2021
entrez: 12 11 2020
Statut: ppublish

Résumé

TNF receptor-associated periodic syndrome (TRAPS) is a rare autoinflammatory disease caused by dominant mutation of the TNF super family receptor 1A (TNFRSF1A) gene. Data regarding long-term treatment outcomes are lacking. To assess correlations of genotype-phenotypes in patients with TRAPS, as defined by the International Study Group for Systemic Autoinflammatory Diseases (INSAID) classification and Eurofever criteria, with treatment responses. Data from 226 patients with variants of the TNFRSF1A gene and enrolled in the Eurofever registry were classified according to the INSAID classification in groups A (pathogenic or likely pathogenic variants), B (variants of uncertain significance or not classified variants), and C (benign or likely benign variants) and screened for Eurofever criteria. In group A (127 of 226 patients, 56%), all fulfilled Eurofever criteria and 20 of 127 patients (16%) developed AA amyloidosis. In group B (78 of 226 patients, 35%), 40 of 78 patients (51%) did not fulfill Eurofever criteria, displaying a lower incidence of abdominal pain (P < .02) and higher efficacy rate of on-demand nonsteroidal anti-inflammatory drugs (P < .02) and colchicine (P < .001). Group C (21 of 226 patients, 9%) presented a milder disease (P < .02) and none fulfilled Eurofever criteria. Anti-IL-1 drugs were the most frequently used in patients fulfilling Eurofever criteria, with the highest efficacy rate (>85% complete response). No patients on anti-IL-1 treatments developed AA amyloidosis, and 7 women with a history of failure to conceive had successful pregnancies. Anti-IL-1 drugs are the best maintenance treatment in patients with TRAPS. The diagnosis of TRAPS should be considered very carefully in patients of group B not fulfilling Eurofever criteria and group C, and colchicine may be preferable as the first maintenance treatment.

Sections du résumé

BACKGROUND
TNF receptor-associated periodic syndrome (TRAPS) is a rare autoinflammatory disease caused by dominant mutation of the TNF super family receptor 1A (TNFRSF1A) gene. Data regarding long-term treatment outcomes are lacking.
OBJECTIVE
To assess correlations of genotype-phenotypes in patients with TRAPS, as defined by the International Study Group for Systemic Autoinflammatory Diseases (INSAID) classification and Eurofever criteria, with treatment responses.
METHODS
Data from 226 patients with variants of the TNFRSF1A gene and enrolled in the Eurofever registry were classified according to the INSAID classification in groups A (pathogenic or likely pathogenic variants), B (variants of uncertain significance or not classified variants), and C (benign or likely benign variants) and screened for Eurofever criteria.
RESULTS
In group A (127 of 226 patients, 56%), all fulfilled Eurofever criteria and 20 of 127 patients (16%) developed AA amyloidosis. In group B (78 of 226 patients, 35%), 40 of 78 patients (51%) did not fulfill Eurofever criteria, displaying a lower incidence of abdominal pain (P < .02) and higher efficacy rate of on-demand nonsteroidal anti-inflammatory drugs (P < .02) and colchicine (P < .001). Group C (21 of 226 patients, 9%) presented a milder disease (P < .02) and none fulfilled Eurofever criteria. Anti-IL-1 drugs were the most frequently used in patients fulfilling Eurofever criteria, with the highest efficacy rate (>85% complete response). No patients on anti-IL-1 treatments developed AA amyloidosis, and 7 women with a history of failure to conceive had successful pregnancies.
CONCLUSION
Anti-IL-1 drugs are the best maintenance treatment in patients with TRAPS. The diagnosis of TRAPS should be considered very carefully in patients of group B not fulfilling Eurofever criteria and group C, and colchicine may be preferable as the first maintenance treatment.

Identifiants

pubmed: 33181346
pii: S2213-2198(20)31208-3
doi: 10.1016/j.jaip.2020.10.053
pii:
doi:

Substances chimiques

Colchicine SML2Y3J35T

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

783-791.e4

Informations de copyright

Copyright © 2020 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

Auteurs

Riccardo Papa (R)

Centre for Autoinflammatory Diseases and Immunodeficiencies, IRCCS Istituto Giannina Gaslini, Genoa, Italy.

Thirusha Lane (T)

National Amyloidosis Centre, UCL Division of Medicine, Royal Free Campus, University College London, London, United Kingdom.

Kirsten Minden (K)

Charité University Hospital Berlin and German Rheumatism Research Center, Berlin, Germany.

Isabelle Touitou (I)

Department of Medical Genetics, Rare Diseases and Personalized Medicine, Reference Center CEREMAIA, CHU Montpellier, INSERM, University of Montpellier, Montpellier, France.

Luca Cantarini (L)

Rheumatology Unit, Policlinico le Scotte, University of Siena, Siena, Italy.

Marco Cattalini (M)

Clinica Pediatrica, Spedali Civili, University of Brescia, Brescia, Italy.

Laura Obici (L)

Amyloidosis Research and Treatment Centre, Fondazione IRCCS Policlinico San Matteo, Pavia, Italy.

Annette F Jansson (AF)

Department of Pediatrics, Dr. von Hauner Children's Hospital, LMU Munich, Munich, Germany.

Alexandre Belot (A)

Pediatric Nephrology, Rheumatology, Dermatology Unit, National Reference Centre for Rheumatic and AutoImmune disease in childrEn (RAISE), Hopital Femme-Mère Enfant, Groupement Hospitalier Est, Lyon, France.

Joost Frenkel (J)

Department of Pediatric Immunology and Rheumatology, Wilhelmina Children's Hospital, University Medical Center Utrecht, Utrecht, the Netherlands.

Jordi Anton (J)

Unidad de Reumatología Pediátrica, Hospital Sant Joan de Déu, Universitat de Barcelona, Esplugues de Llobregat (Barcelona), Spain.

Beata Wolska-Kusnierz (B)

Department of Immunology, Children's Memorial Health Institute, Warsaw, Poland.

Rainer Berendes (R)

Pediatric Rheumatology, Kinderkrankenhaus St. Marien, Landshut, Germany.

Agustin Remesal (A)

Pediatric Rheumatology Unit, Hospital Universitario La Paz, Madrid, Spain.

Marija Jelusic (M)

University of Zagreb School of Medicine, University Hospital Centre Zagreb, Zagreb, Croatia.

Esther Hoppenreijs (E)

Department of Pediatric Rheumatology, Radboud UMC, Nijmegen, the Netherlands.

Graciela Espada (G)

Division of Rheumatology, Hospital de Niños Ricardo Gutierrez, Buenos Aires, Argentina.

Irina Nikishina (I)

Pediatric Department, V.A. Nasonova Research Institute of Rheumatology, Moscow, Russian Federation.

Maria Cristina Maggio (MC)

Dipartimento Universitario PROMISE "G. D'Alessandro," University of Palermo, Palermo, Italy.

Francesca Bovis (F)

Department of Health Science, University of Genoa, Genoa, Italy.

Marta Masini (M)

Department of Health Science, University of Genoa, Genoa, Italy.

Taryn Youngstein (T)

National Amyloidosis Centre, UCL Division of Medicine, Royal Free Campus, University College London, London, United Kingdom.

Tamer Rezk (T)

National Amyloidosis Centre, UCL Division of Medicine, Royal Free Campus, University College London, London, United Kingdom.

Charalampia Papadopoulou (C)

Department of Infection, Inflammation and Rheumatology, UCL Great Ormond Street Institute of Child Health, London, United Kingdom.

Paul A Brogan (PA)

Department of Infection, Inflammation and Rheumatology, UCL Great Ormond Street Institute of Child Health, London, United Kingdom.

Philip N Hawkins (PN)

National Amyloidosis Centre, UCL Division of Medicine, Royal Free Campus, University College London, London, United Kingdom.

Patricia Woo (P)

Department of Infection, Inflammation and Rheumatology, UCL Great Ormond Street Institute of Child Health, London, United Kingdom.

Nicolino Ruperto (N)

UOSID Centro Trial, IRCCS Istituto Giannina Gaslini, Genoa, Italy.

Marco Gattorno (M)

Centre for Autoinflammatory Diseases and Immunodeficiencies, IRCCS Istituto Giannina Gaslini, Genoa, Italy. Electronic address: marcogattorno@gaslini.org.

Helen J Lachmann (HJ)

National Amyloidosis Centre, UCL Division of Medicine, Royal Free Campus, University College London, London, United Kingdom. Electronic address: h.lachmann@ucl.ac.uk.

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