B-cell
B10-cell
EAE
ERK1/2
demyelination
oligodendrocyte
Journal
ASN neuro
ISSN: 1759-0914
Titre abrégé: ASN Neuro
Pays: United States
ID NLM: 101507115
Informations de publication
Date de publication:
Historique:
entrez:
24
11
2020
pubmed:
25
11
2020
medline:
7
10
2021
Statut:
ppublish
Résumé
The ERK1/2 signaling pathway promotes myelin wrapping during development and remyelination, and sustained ERK1/2 activation in the oligodendrocyte (OL) lineage results in hypermyelination of the CNS. We therefore hypothesized that increased ERK1/2 signaling in the OL lineage would 1) protect against immune-mediated demyelination due to increased baseline myelin thickness and/or 2) promote enhanced remyelination and thus functional recovery after experimental autoimmune encephalomyelitis (EAE) induction.
Identifiants
pubmed: 33228381
doi: 10.1177/1759091420971916
pmc: PMC7691909
doi:
Substances chimiques
2',3'-Cyclic Nucleotide 3'-Phosphodiesterase
EC 3.1.4.37
Cnp protein, mouse
EC 3.1.4.37
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
1759091420971916Subventions
Organisme : NINDS NIH HHS
ID : R01 NS082203
Pays : United States
Organisme : NINDS NIH HHS
ID : R37 NS082203
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS091084
Pays : United States
Organisme : NINDS NIH HHS
ID : F31 NS108521
Pays : United States
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