Adverse effects of Z-drugs for sleep disturbance in people living with dementia: a population-based cohort study.


Journal

BMC medicine
ISSN: 1741-7015
Titre abrégé: BMC Med
Pays: England
ID NLM: 101190723

Informations de publication

Date de publication:
24 11 2020
Historique:
received: 14 07 2020
accepted: 20 10 2020
entrez: 24 11 2020
pubmed: 25 11 2020
medline: 23 2 2021
Statut: epublish

Résumé

Sleep disturbance is common in dementia and often treated with Z-drugs (zopiclone, zaleplon, and zolpidem). While some observational studies suggest that Z-drugs are associated with adverse events such as falls and fracture risks in older people, this has not been studied in dementia. We used data from 27,090 patients diagnosed with dementia between January 2000 and March 2016 from the Clinical Practice Research Datalink linked to Hospital Episodes Statistics data in England. We compared adverse events for 3532 patients newly prescribed Z-drugs by time-varying dosage to (1) 1833 non-sedative-users with sleep disturbance; (2) 10,214 non-sedative-users with proximal GP consultation matched on age, sex, and antipsychotic use; and (3) 5172 patients newly prescribed benzodiazepines. We defined higher dose Z-drugs and benzodiazepines as prescriptions equivalent to ≥ 7.5 mg zopiclone or > 5 mg diazepam daily. Cox regression was used to estimate hazard ratios (HRs) for incident fracture, hip fracture, fall, mortality, acute bacterial infection, ischaemic stroke/transient ischaemic attack, and venous thromboembolism over a 2-year follow-up, adjusted for demographic- and health-related covariates. The mean (SD) age of patients was 83 (7.7) years, and 16,802 (62%) were women. Of 3532 patients prescribed Z-drugs, 584 (17%) were initiated at higher doses. For patients prescribed higher dose Z-drugs relative to non-users with sleep disturbance, the HRs (95% confidence interval) for fractures, hip fractures, falls, and ischaemic stroke were 1.67 (1.13-2.46), 1.96 (1.16-3.31), 1.33 (1.06-1.66), and 1.88 (1.14-3.10), respectively. We observed similar associations when compared to non-sedative-users with proximal GP consultation. Minimal or inconsistent excess risks were observed at ≤ 3.75 mg zopiclone or equivalent daily, and for mortality, infection, and venous thromboembolism. We observed no differences in adverse events for Z-drugs compared to benzodiazepines, except lower mortality rates with Z-drugs (HR [95% confidence interval] of 0.73 [0.64-0.83]). Higher dose Z-drug use in dementia is associated with increased fracture and stroke risks, similar or greater to that for higher dose benzodiazepines. Higher dose Z-drugs should be avoided, if possible, in people living with dementia, and non-pharmacological alternatives preferentially considered. Prescriptions for higher dose Z-drugs in dementia should be regularly reviewed. ENCePP e-register of studies, EUPAS18006.

Sections du résumé

BACKGROUND
Sleep disturbance is common in dementia and often treated with Z-drugs (zopiclone, zaleplon, and zolpidem). While some observational studies suggest that Z-drugs are associated with adverse events such as falls and fracture risks in older people, this has not been studied in dementia.
METHODS
We used data from 27,090 patients diagnosed with dementia between January 2000 and March 2016 from the Clinical Practice Research Datalink linked to Hospital Episodes Statistics data in England. We compared adverse events for 3532 patients newly prescribed Z-drugs by time-varying dosage to (1) 1833 non-sedative-users with sleep disturbance; (2) 10,214 non-sedative-users with proximal GP consultation matched on age, sex, and antipsychotic use; and (3) 5172 patients newly prescribed benzodiazepines. We defined higher dose Z-drugs and benzodiazepines as prescriptions equivalent to ≥ 7.5 mg zopiclone or > 5 mg diazepam daily. Cox regression was used to estimate hazard ratios (HRs) for incident fracture, hip fracture, fall, mortality, acute bacterial infection, ischaemic stroke/transient ischaemic attack, and venous thromboembolism over a 2-year follow-up, adjusted for demographic- and health-related covariates.
RESULTS
The mean (SD) age of patients was 83 (7.7) years, and 16,802 (62%) were women. Of 3532 patients prescribed Z-drugs, 584 (17%) were initiated at higher doses. For patients prescribed higher dose Z-drugs relative to non-users with sleep disturbance, the HRs (95% confidence interval) for fractures, hip fractures, falls, and ischaemic stroke were 1.67 (1.13-2.46), 1.96 (1.16-3.31), 1.33 (1.06-1.66), and 1.88 (1.14-3.10), respectively. We observed similar associations when compared to non-sedative-users with proximal GP consultation. Minimal or inconsistent excess risks were observed at ≤ 3.75 mg zopiclone or equivalent daily, and for mortality, infection, and venous thromboembolism. We observed no differences in adverse events for Z-drugs compared to benzodiazepines, except lower mortality rates with Z-drugs (HR [95% confidence interval] of 0.73 [0.64-0.83]).
CONCLUSIONS
Higher dose Z-drug use in dementia is associated with increased fracture and stroke risks, similar or greater to that for higher dose benzodiazepines. Higher dose Z-drugs should be avoided, if possible, in people living with dementia, and non-pharmacological alternatives preferentially considered. Prescriptions for higher dose Z-drugs in dementia should be regularly reviewed.
TRIAL REGISTRATION
ENCePP e-register of studies, EUPAS18006.

Identifiants

pubmed: 33228664
doi: 10.1186/s12916-020-01821-5
pii: 10.1186/s12916-020-01821-5
pmc: PMC7683259
doi:

Substances chimiques

Acetamides 0
Azabicyclo Compounds 0
Piperazines 0
Pyrimidines 0
zopiclone 03A5ORL08Q
Zolpidem 7K383OQI23
zaleplon S62U433RMH

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

351

Subventions

Organisme : Department of Health
ID : 14/221/02
Pays : United Kingdom
Organisme : Health Technology Assessment Programme
ID : 14/221/02
Pays : International

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Auteurs

Kathryn Richardson (K)

Norwich Medical School, University of East Anglia, Norwich, NR4 7TJ, UK. kathryn.richardson@uea.ac.uk.

Yoon K Loke (YK)

Norwich Medical School, University of East Anglia, Norwich, NR4 7TJ, UK.

Chris Fox (C)

Norwich Medical School, University of East Anglia, Norwich, NR4 7TJ, UK.

Ian Maidment (I)

School of Life and Health Sciences, Aston University, Birmingham, B4 7ET, UK.

Robert Howard (R)

Division of Psychiatry, UCL Division of Psychiatry, University College London, Maple House, 149 Tottenham Court Road, London, W1T 7NF, UK.

Nicholas Steel (N)

Norwich Medical School, University of East Anglia, Norwich, NR4 7TJ, UK.

Antony Arthur (A)

School of Health Sciences, University of East Anglia, Norwich, NR4 7TJ, UK.

Penelope J Boyd (PJ)

Norwich Medical School, University of East Anglia, Norwich, NR4 7TJ, UK.

Clare Aldus (C)

School of Health Sciences, University of East Anglia, Norwich, NR4 7TJ, UK.

Clive Ballard (C)

Medical School, University of Exeter, Exeter, EX1 2LU, UK.

George M Savva (GM)

School of Health Sciences, University of East Anglia, Norwich, NR4 7TJ, UK.
Quadram Institute Bioscience, Norwich Research Park, Norwich, NR4 7UQ, UK.

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