Absence of CD36 alters systemic vitamin A homeostasis.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
23 11 2020
Historique:
received: 27 07 2020
accepted: 03 11 2020
entrez: 24 11 2020
pubmed: 25 11 2020
medline: 17 4 2021
Statut: epublish

Résumé

Fatty acid translocase (CD36) is a scavenger receptor with multiple ligands and diverse physiological actions. We recently reported that alcohol-induced hepatic retinoid mobilization is impaired in Cd36

Identifiants

pubmed: 33230291
doi: 10.1038/s41598-020-77411-5
pii: 10.1038/s41598-020-77411-5
pmc: PMC7683526
doi:

Substances chimiques

CD36 Antigens 0
Cd36 protein, mouse 0
Chylomicrons 0
CrbpI protein, mouse 0
Receptors, Retinoic Acid 0
Retinol-Binding Proteins, Cellular 0
Retinyl Esters 0
retinoic acid receptor beta 0
Vitamin A 11103-57-4
Cyp26a1 protein, mouse EC 1.14.14.1
Retinoic Acid 4-Hydroxylase EC 1.14.14.1
Acyltransferases EC 2.3.-
lecithin-retinol acyltransferase EC 2.3.1.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

20386

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Auteurs

Michael J Trites (MJ)

Department of Physiology, University of Alberta, 7-49 Medical Sciences Building, Edmonton, AB, T6G 2H7, Canada.
Group on the Molecular and Cell Biology of Lipids, Faculty of Medicine and Dentistry, University of Alberta, Edmonton, AB, Canada.

Maria Febbraio (M)

Department of Dentistry, Faculty of Medicine and Dentistry, University of Alberta, Edmonton, AB, Canada.

Robin D Clugston (RD)

Department of Physiology, University of Alberta, 7-49 Medical Sciences Building, Edmonton, AB, T6G 2H7, Canada. clugston@ualberta.ca.
Group on the Molecular and Cell Biology of Lipids, Faculty of Medicine and Dentistry, University of Alberta, Edmonton, AB, Canada. clugston@ualberta.ca.

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Classifications MeSH