Association of PARP1-specific polymorphisms and haplotypes with non-small cell lung cancer subtypes.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2020
Historique:
received: 20 04 2020
accepted: 20 11 2020
entrez: 7 12 2020
pubmed: 8 12 2020
medline: 29 1 2021
Statut: epublish

Résumé

The carcinogenesis role of PARP1 in lung cancer is still not clear. Analysis at allelic levels cannot fully explain the function of PARP1 on lung cancer. Our study aims to further explore the relation between PARP1 haplotypes and lung cancer. DNA and RNA were extracted from non-small cell lung cancer (NSCLC) tumor and adjacent normal fresh frozen tissue. Five PARP1-SNPs were genotyped and PARP1-specific SNPs were imputed using IMPUTE and SHAPEIT software. The SNPs were subjected to allelic, haplotype and SNP-SNP interaction analyses. Correlation between SNPs and mRNA/protein expressions were performed. SNP imputation inferred the ungenotyped SNPs and increased the power for association analysis. Tumor tissue samples are more likely to carry rs1805414 (OR = 1.85; 95% CI: 1.12-3.06; P-value: 0.017) and rs1805404 (OR = 2.74; 95%CI 1.19-6.32; P-value: 0.015) compared to normal tissues. Our study is the first study to show that haplotypes comprising of 5 SNPs on PARP1 (rs1136410, rs3219073, rs1805414, rs1805404, rs1805415) is able to differentiate the NSCLC tumor from normal tissues. Interaction between rs3219073, rs1805415, and rs1805414 were significantly associated with the NSCLC tumor with OR ranging from 3.61-6.75; 95%CI from 1.82 to 19.9; P-value<0.001. PARP1 haplotypes may serve as a better predictor in lung cancer development and prognosis compared to single alleles.

Identifiants

pubmed: 33284833
doi: 10.1371/journal.pone.0243509
pii: PONE-D-20-11416
pmc: PMC7721167
doi:

Substances chimiques

PARP1 protein, human EC 2.4.2.30
Poly (ADP-Ribose) Polymerase-1 EC 2.4.2.30

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0243509

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Jing Jin (J)

Department of Epidemiology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.

Heather Robeson (H)

Department of Epidemiology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.

Pebbles Fagan (P)

Department of Health Behavior and Health Education, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Winthrop P. Rockefeller Cancer Institute, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Center for the Studies of Tobacco, Fay W. Boozman College of Public Health, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.

Mohammed S Orloff (MS)

Department of Epidemiology, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Winthrop P. Rockefeller Cancer Institute, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.
Center for the Studies of Tobacco, Fay W. Boozman College of Public Health, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States of America.

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Classifications MeSH