Association between ADAMTS13 activity-VWF antigen imbalance and the therapeutic effect of HAIC in patients with hepatocellular carcinoma.


Journal

World journal of gastroenterology
ISSN: 2219-2840
Titre abrégé: World J Gastroenterol
Pays: United States
ID NLM: 100883448

Informations de publication

Date de publication:
07 Dec 2020
Historique:
received: 04 08 2020
revised: 09 10 2020
accepted: 13 11 2020
entrez: 28 12 2020
pubmed: 29 12 2020
medline: 15 5 2021
Statut: ppublish

Résumé

Prediction of HAIC treatment response is important for improving the prognosis in patients with hepatocellular carcinoma (HCC). The progression of HCC is related to hypercoagulability and angiogenesis. It is known that ADAMTS13 and von Willebrand factor (VWF) are related to hypercoagulability. In addition, previous study reported that the association between ADAMTS13 and VWF, and angiogenesis To investigate whether ADAMTS13 and VWF become useful biomarkers of treatment response in HCC patients before the initiation of HAIC treatment. Seventy-two patients were enrolled in this study. ADAMTS13 activity (ADAMTS13:AC), VWF antigen (VWF:Ag) and VEGF levels were determined ADAMTS13:AC levels in HCC patients with stable disease (SD) + partial response (PR) of HAIC treatment were significantly higher than those with progressive disease (PD) ( VWF:Ag/ADAMTS13:AC ratio may become a useful biomarker of treatment response in HCC patients before the initiation of HAIC treatment.

Sections du résumé

BACKGROUND BACKGROUND
Prediction of HAIC treatment response is important for improving the prognosis in patients with hepatocellular carcinoma (HCC). The progression of HCC is related to hypercoagulability and angiogenesis. It is known that ADAMTS13 and von Willebrand factor (VWF) are related to hypercoagulability. In addition, previous study reported that the association between ADAMTS13 and VWF, and angiogenesis
AIM OBJECTIVE
To investigate whether ADAMTS13 and VWF become useful biomarkers of treatment response in HCC patients before the initiation of HAIC treatment.
METHODS METHODS
Seventy-two patients were enrolled in this study. ADAMTS13 activity (ADAMTS13:AC), VWF antigen (VWF:Ag) and VEGF levels were determined
RESULTS RESULTS
ADAMTS13:AC levels in HCC patients with stable disease (SD) + partial response (PR) of HAIC treatment were significantly higher than those with progressive disease (PD) (
CONCLUSION CONCLUSIONS
VWF:Ag/ADAMTS13:AC ratio may become a useful biomarker of treatment response in HCC patients before the initiation of HAIC treatment.

Identifiants

pubmed: 33362379
doi: 10.3748/wjg.v26.i45.7232
pmc: PMC7723670
doi:

Substances chimiques

Vascular Endothelial Growth Factor A 0
von Willebrand Factor 0
ADAMTS13 Protein EC 3.4.24.87
ADAMTS13 protein, human EC 3.4.24.87

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

7232-7241

Informations de copyright

©The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved.

Déclaration de conflit d'intérêts

Conflict-of-interest statement: The authors declare that they have no conflicts of interest.

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Auteurs

Hiroaki Takaya (H)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan. htky@naramed-u.ac.jp.

Tadashi Namisaki (T)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Kei Moriya (K)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Naotaka Shimozato (N)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Kosuke Kaji (K)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Hiroyuki Ogawa (H)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Koji Ishida (K)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Yuki Tsuji (Y)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Daisuke Kaya (D)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Yukihisa Fujinaga (Y)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Norihisa Nishimura (N)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Yasuhiko Sawada (Y)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Hideto Kawaratani (H)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Takemi Akahane (T)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Masanori Matsumoto (M)

Department of Blood Transfusion Medicine, Nara Medical University, Kashihara, Nara 634-8522, Japan.

Hitoshi Yoshiji (H)

Department of Gastroenterology, Nara Medical University, Kashihara, Nara 634-8522, Japan.

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