Association of an increase in serum albumin levels with positive 1-year outcomes in acute decompensated heart failure: A cohort study.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2020
Historique:
received: 25 09 2020
accepted: 27 11 2020
entrez: 28 12 2020
pubmed: 29 12 2020
medline: 4 2 2021
Statut: epublish

Résumé

Despite the prognostic importance of hypoalbuminemia, the prognostic implication of a change in albumin levels has not been fully investigated during hospitalization in patients with acute decompensated heart failure (ADHF). Using the data from the Kyoto Congestive Heart Failure registry on 3160 patients who were discharged alive for acute heart failure hospitalization and in whom the change in albumin levels was calculated at discharge, we evaluated the association with an increase in serum albumin levels from admission to discharge and clinical outcomes by a multivariable Cox hazard model. The primary outcome measure was a composite of all-cause death or hospitalization for heart failure. Patients with increased albumin levels (N = 1083, 34.3%) were younger and less often had smaller body mass index and renal dysfunction than those with no increase in albumin levels (N = 2077, 65.7%). Median follow-up was 475 days with a 96% 1-year follow-up rate. Relative to the group with no increase in albumin levels, the lower risk of the increased albumin group remained significant for the primary outcome measure (hazard ratio: 0.78, 95% confidence interval: 0.69-0.90: P = 0.0004) after adjusting for confounders including baseline albumin levels. When stratified by the quartiles of baseline albumin levels, the favorable effect of increased albumin was more pronounced in the lower quartiles of albumin levels, but without a significant interaction effect (interaction P = 0.49). Independent of baseline albumin levels, an increase in albumin during index hospitalization was associated with a lower 1-year risk for a composite of all-cause death and hospitalization in patients with acute heart failure.

Sections du résumé

BACKGROUND
Despite the prognostic importance of hypoalbuminemia, the prognostic implication of a change in albumin levels has not been fully investigated during hospitalization in patients with acute decompensated heart failure (ADHF).
METHODS
Using the data from the Kyoto Congestive Heart Failure registry on 3160 patients who were discharged alive for acute heart failure hospitalization and in whom the change in albumin levels was calculated at discharge, we evaluated the association with an increase in serum albumin levels from admission to discharge and clinical outcomes by a multivariable Cox hazard model. The primary outcome measure was a composite of all-cause death or hospitalization for heart failure.
FINDINGS
Patients with increased albumin levels (N = 1083, 34.3%) were younger and less often had smaller body mass index and renal dysfunction than those with no increase in albumin levels (N = 2077, 65.7%). Median follow-up was 475 days with a 96% 1-year follow-up rate. Relative to the group with no increase in albumin levels, the lower risk of the increased albumin group remained significant for the primary outcome measure (hazard ratio: 0.78, 95% confidence interval: 0.69-0.90: P = 0.0004) after adjusting for confounders including baseline albumin levels. When stratified by the quartiles of baseline albumin levels, the favorable effect of increased albumin was more pronounced in the lower quartiles of albumin levels, but without a significant interaction effect (interaction P = 0.49).
CONCLUSIONS
Independent of baseline albumin levels, an increase in albumin during index hospitalization was associated with a lower 1-year risk for a composite of all-cause death and hospitalization in patients with acute heart failure.

Identifiants

pubmed: 33370299
doi: 10.1371/journal.pone.0243818
pii: PONE-D-20-30330
pmc: PMC7769473
doi:

Substances chimiques

Serum Albumin 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0243818

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

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Auteurs

Takao Kato (T)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Hidenori Yaku (H)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Takeshi Morimoto (T)

Department of Clinical Epidemiology, Hyogo College of Medicine, Hyogo, Japan.

Yasutaka Inuzuka (Y)

Department of Cardiovascular Medicine, Shiga General Hospital, Shiga, Japan.

Yodo Tamaki (Y)

Division of Cardiology, Tenri Hospital, Nara, Japan.

Neiko Ozasa (N)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Erika Yamamoto (E)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Yusuke Yoshikawa (Y)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Takeshi Kitai (T)

Department of Cardiovascular Medicine, Kobe City Medical Center General Hospital, Hyogo, Japan.

Ryoji Taniguchi (R)

Department of Cardiology, Hyogo Prefectural Amagasaki General Medical Center, Hyogo, Japan.

Moritake Iguchi (M)

Department of Cardiology, National Hospital Organization Kyoto Medical Center, Kyoto, Japan.

Masashi Kato (M)

Department of Cardiology, Mitsubishi Kyoto Hospital, Kyoto, Japan.

Mamoru Takahashi (M)

Department of Cardiology, Shimabara Hospital, Kyoto, Japan.

Toshikazu Jinnai (T)

Department of Cardiology, Japanese Red Cross Otsu Hospital, Shiga, Japan.

Tomoyuki Ikeda (T)

Department of Cardiology, Hikone Municipal Hospital, Shiga, Japan.

Kazuya Nagao (K)

Department of Cardiology, Osaka Red Cross Hospital, Osaka, Japan.

Takafumi Kawai (T)

Department of Cardiology, Kishiwada City Hospital, Osaka, Japan.

Akihiro Komasa (A)

Department of Cardiology, Kansai Electric Power Hospital, Osaka, Japan.

Ryusuke Nishikawa (R)

Department of Cardiology, Shizuoka General Hospital, Shizuoka, Japan.

Yuichi Kawase (Y)

Department of Cardiology, Kurashiki Central Hospital, Okayama, Japan.

Takashi Morinaga (T)

Department of Cardiology, Kokura Memorial Hospital, Fukuoka, Japan.

Mitsunori Kawato (M)

Department of Cardiology, Kobe City Nishi-Kobe Medical Center, Hyogo, Japan.

Yuta Seko (Y)

Kitano Hospital, Osaka, Japan.

Masayuki Shiba (M)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Mamoru Toyofuku (M)

Department of Cardiology, Japanese Red Cross Wakayama Medical Center, Wakayama, Japan.

Yutaka Furukawa (Y)

Department of Cardiovascular Medicine, Kobe City Medical Center General Hospital, Hyogo, Japan.

Kenji Ando (K)

Department of Cardiology, Kokura Memorial Hospital, Fukuoka, Japan.

Kazushige Kadota (K)

Department of Cardiology, Kurashiki Central Hospital, Okayama, Japan.

Yukihito Sato (Y)

Department of Cardiology, Hyogo Prefectural Amagasaki General Medical Center, Hyogo, Japan.

Koichiro Kuwahara (K)

Department of Cardiovascular Medicine, Shinshu University Graduate School of Medicine, Matsumoto, Japan.

Takeshi Kimura (T)

Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine, Kyoto, Japan.

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