N-Terminal Pro-B-Type Natriuretic Peptide and Longitudinal Risk of Hypertension.


Journal

American journal of hypertension
ISSN: 1941-7225
Titre abrégé: Am J Hypertens
Pays: United States
ID NLM: 8803676

Informations de publication

Date de publication:
22 05 2021
Historique:
received: 01 05 2020
revised: 21 08 2020
accepted: 23 12 2020
pubmed: 31 12 2020
medline: 15 12 2021
entrez: 30 12 2020
Statut: ppublish

Résumé

Hypertension is a common condition that increases risk for future cardiovascular disease. N-terminal B-type natriuretic peptide (NT-proBNP) is higher in individuals with hypertension, but studies of its association with hypertension risk have been mixed. The REasons for Geographic And Racial Differences in Stroke (REGARDS) study enrolled 30,239 U.S. Black or White adults aged ≥45 years from 2003 to 2007. A subcohort included 4,400 participants who completed a second assessment in 2013-2016. NT-proBNP was measured by immunoassay in 1,323 participants without baseline hypertension, defined as blood pressure ≥140/90 or self-reported antihypertensive prescriptions. Two robust Poisson regression models assessed hypertension risk, yielding incidence rate ratios (IRRs): Model 1 included behavioral and demographic covariates and Model 2 added risk factors. A sensitivity analysis using a less conservative definition of hypertension (blood pressure ≥130/80 or self-reported antihypertensive prescriptions) was conducted. Four hundred and sixty-six participants developed hypertension after mean follow-up of 9.4 years. NT-proBNP was not associated with hypertension (Model 2 IRR per SD log NT-proBNP 1.01, 95% confidence interval 0.92-1.12), with no differences by sex, body mass index, age, or race. Similar findings were seen in lower-threshold sensitivity analysis. NT-proBNP was not associated with incident hypertension in REGARDS; this did not differ by race or sex.

Sections du résumé

BACKGROUND
Hypertension is a common condition that increases risk for future cardiovascular disease. N-terminal B-type natriuretic peptide (NT-proBNP) is higher in individuals with hypertension, but studies of its association with hypertension risk have been mixed.
METHODS
The REasons for Geographic And Racial Differences in Stroke (REGARDS) study enrolled 30,239 U.S. Black or White adults aged ≥45 years from 2003 to 2007. A subcohort included 4,400 participants who completed a second assessment in 2013-2016. NT-proBNP was measured by immunoassay in 1,323 participants without baseline hypertension, defined as blood pressure ≥140/90 or self-reported antihypertensive prescriptions. Two robust Poisson regression models assessed hypertension risk, yielding incidence rate ratios (IRRs): Model 1 included behavioral and demographic covariates and Model 2 added risk factors. A sensitivity analysis using a less conservative definition of hypertension (blood pressure ≥130/80 or self-reported antihypertensive prescriptions) was conducted.
RESULTS
Four hundred and sixty-six participants developed hypertension after mean follow-up of 9.4 years. NT-proBNP was not associated with hypertension (Model 2 IRR per SD log NT-proBNP 1.01, 95% confidence interval 0.92-1.12), with no differences by sex, body mass index, age, or race. Similar findings were seen in lower-threshold sensitivity analysis.
CONCLUSIONS
NT-proBNP was not associated with incident hypertension in REGARDS; this did not differ by race or sex.

Identifiants

pubmed: 33378421
pii: 6055606
doi: 10.1093/ajh/hpaa224
pmc: PMC8140656
doi:

Substances chimiques

Peptide Fragments 0
pro-brain natriuretic peptide (1-76) 0
Natriuretic Peptide, Brain 114471-18-0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

476-483

Subventions

Organisme : NINDS NIH HHS
ID : U01 NS041588
Pays : United States

Informations de copyright

© American Journal of Hypertension, Ltd 2020. All rights reserved. For Permissions, please email: journals.permissions@oup.com.

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Auteurs

Charles D Nicoli (CD)

University of Vermont Larner College of Medicine, Burlington, Vermont, USA.

Timothy B Plante (TB)

Department of Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.

D Leann Long (DL)

Department of Biostatistics, University of Alabama at Birmingham, Birmingham, Alabama, USA.

Suzanne E Judd (SE)

Department of Biostatistics, University of Alabama at Birmingham, Birmingham, Alabama, USA.

Leslie A McClure (LA)

Department of Epidemiology and Biostatistics, School of Public Health, Drexel University, Philadelphia, Pennsylvania, USA.

Pankaj Arora (P)

Division of Cardiovascular Disease, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama, USA.

Mary Cushman (M)

Department of Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.
Department of Pathology and Laboratory Medicine, University of Vermont Larner College of Medicine, Burlington, Vermont, USA.

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