Effects of hypoxia-inducible factor-1α and hypoxia-inducible factor-2α overexpression on hepatocellular carcinoma survival: A systematic review with meta-analysis.


Journal

Journal of gastroenterology and hepatology
ISSN: 1440-1746
Titre abrégé: J Gastroenterol Hepatol
Pays: Australia
ID NLM: 8607909

Informations de publication

Date de publication:
Jun 2021
Historique:
revised: 08 12 2020
received: 24 07 2020
accepted: 28 12 2020
pubmed: 5 1 2021
medline: 3 11 2021
entrez: 4 1 2021
Statut: ppublish

Résumé

The role of hypoxia-inducible factor-1α (HIF-1α) and hypoxia-inducible factor-2α (HIF-2α) has been implicated in the clinical prognosis of hepatocellular carcinoma (HCC), but the results remain controversial. We aim to investigate the association of HIF-1α and HIF-2α overexpression with the prognosis and clinicopathological features of HCC. A systematic search was conducted in PubMed, Embase, Scopus, Web of Science, and Cochrane Library until June 20, 2020. Meta-analysis was conducted to generate combined HRs with 95% confidence intervals (CI) for overall survival (OS) and disease-free survival (DFS). Odds ratios (ORs) with 95% CI were also derived by fixed or random effect model. Twenty-two studies involving 3238 patients were included. Combined data suggested that overexpression of HIF-1α in HCC was not only correlated with poorer OS [HR = 1.75 (95% CI: 1.53-2.00)] and DFS [HR = 1.64 (95% CI: 1.34-2.00)] but was also positively associated with vascular invasion [OR = 1.83 (95% CI: 1.36-2.48)], tumor size [OR = 1.36 (95% CI: 1.12-1.66)], and tumor number [1.74 (95% CI: 1.34-2.25)]. In contrast, HIF-2α overexpression was not associated with the prognosis and clinicopathological features of HCC. Our data provided compelling evidence of a worse prognosis of HCC in HIF-1α overexpression patients but not HIF-2α overexpression ones.

Sections du résumé

BACKGROUND AND AIM OBJECTIVE
The role of hypoxia-inducible factor-1α (HIF-1α) and hypoxia-inducible factor-2α (HIF-2α) has been implicated in the clinical prognosis of hepatocellular carcinoma (HCC), but the results remain controversial. We aim to investigate the association of HIF-1α and HIF-2α overexpression with the prognosis and clinicopathological features of HCC.
METHODS METHODS
A systematic search was conducted in PubMed, Embase, Scopus, Web of Science, and Cochrane Library until June 20, 2020. Meta-analysis was conducted to generate combined HRs with 95% confidence intervals (CI) for overall survival (OS) and disease-free survival (DFS). Odds ratios (ORs) with 95% CI were also derived by fixed or random effect model.
RESULTS RESULTS
Twenty-two studies involving 3238 patients were included. Combined data suggested that overexpression of HIF-1α in HCC was not only correlated with poorer OS [HR = 1.75 (95% CI: 1.53-2.00)] and DFS [HR = 1.64 (95% CI: 1.34-2.00)] but was also positively associated with vascular invasion [OR = 1.83 (95% CI: 1.36-2.48)], tumor size [OR = 1.36 (95% CI: 1.12-1.66)], and tumor number [1.74 (95% CI: 1.34-2.25)]. In contrast, HIF-2α overexpression was not associated with the prognosis and clinicopathological features of HCC.
CONCLUSION CONCLUSIONS
Our data provided compelling evidence of a worse prognosis of HCC in HIF-1α overexpression patients but not HIF-2α overexpression ones.

Identifiants

pubmed: 33393670
doi: 10.1111/jgh.15395
doi:

Substances chimiques

Basic Helix-Loop-Helix Transcription Factors 0
HIF1A protein, human 0
Hypoxia-Inducible Factor 1, alpha Subunit 0
endothelial PAS domain-containing protein 1 1B37H0967P

Types de publication

Journal Article Meta-Analysis Systematic Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1487-1496

Subventions

Organisme : National Natural Science Foundation of China
ID : 81874178
Organisme : National Natural Science Foundation of China
ID : 82073200
Organisme : Taishan Scholars Program for Young Expert of Shandong Province
ID : tsqn20161064

Informations de copyright

© 2021 Journal of Gastroenterology and Hepatology Foundation and John Wiley & Sons Australia, Ltd.

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Auteurs

Zi-Niu Ding (ZN)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Zhao-Ru Dong (ZR)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Zhi-Qiang Chen (ZQ)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Ya-Fei Yang (YF)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Lun-Jie Yan (LJ)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Hai-Chao Li (HC)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Kai-Xuan Liu (KX)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Cheng-Yu Yao (CY)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Yu-Chuan Yan (YC)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Chun-Cheng Yang (CC)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

Tao Li (T)

Department of General Surgery, Qilu Hospital, Shandong University, Jinan, China.

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