Cigarette smoke induces
Adult
Aged
Animals
Arthritis, Experimental
/ pathology
Arthritis, Rheumatoid
/ genetics
Basic Helix-Loop-Helix Transcription Factors
/ metabolism
Cigarette Smoking
/ adverse effects
Female
Humans
Male
Mice
Mice, Inbred C57BL
MicroRNAs
/ genetics
Middle Aged
Osteoclasts
/ metabolism
Osteogenesis
/ drug effects
Receptors, Aryl Hydrocarbon
/ metabolism
Smoke
Th17 Cells
/ drug effects
Tobacco Smoke Pollution
/ adverse effects
Th17
cigarette smoke
exosomes
osteoclastogenesis
rheumatoid arthritis
Journal
Proceedings of the National Academy of Sciences of the United States of America
ISSN: 1091-6490
Titre abrégé: Proc Natl Acad Sci U S A
Pays: United States
ID NLM: 7505876
Informations de publication
Date de publication:
05 01 2021
05 01 2021
Historique:
entrez:
14
1
2021
pubmed:
15
1
2021
medline:
15
5
2021
Statut:
ppublish
Résumé
Rheumatoid arthritis (RA) is a chronic inflammatory disease characterized by joint destruction and severe morbidity. Cigarette smoking (CS) can exacerbate the incidence and severity of RA. Although Th17 cells and the Aryl hydrocarbon receptor (AhR) have been implicated, the mechanism by which CS induces RA development remains unclear. Here, using transcriptomic analysis, we show that
Identifiants
pubmed: 33443169
pii: 2017120118
doi: 10.1073/pnas.2017120118
pmc: PMC7817209
pii:
doi:
Substances chimiques
AHR protein, human
0
Basic Helix-Loop-Helix Transcription Factors
0
MIRN132 microRNA, human
0
MIRN132 microRNA, mouse
0
MicroRNAs
0
Receptors, Aryl Hydrocarbon
0
Smoke
0
Tobacco Smoke Pollution
0
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Commentaires et corrections
Type : CommentIn
Type : ErratumIn
Déclaration de conflit d'intérêts
The authors declare no competing interest.
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