Pretreatment ADC predicts tumor control after Gamma Knife radiosurgery in solid vestibular schwannomas.


Journal

Acta neurochirurgica
ISSN: 0942-0940
Titre abrégé: Acta Neurochir (Wien)
Pays: Austria
ID NLM: 0151000

Informations de publication

Date de publication:
04 2021
Historique:
received: 01 10 2020
accepted: 26 01 2021
pubmed: 4 2 2021
medline: 12 6 2021
entrez: 3 2 2021
Statut: ppublish

Résumé

Radiosurgery is a well-established treatment for vestibular schwannomas (VSs), but it is often difficult to identify which tumors will respond to treatment. We sought to determine whether pretreatment or posttreatment tumor apparent diffusion coefficient (ADC) values could predict tumor control in patients undergoing Gamma Knife radiosurgery (GKRS) and whether these values could differentiate between cases of pseudoprogression and cases of true progression in the early posttreatment period. We retrospectively identified patients who underwent GKRS for solid VSs between June 2008 and November 2016 and who had a minimum follow-up of 36 months. Pretreatment and posttreatment minimum, mean, and maximum ADC values were measured for the whole tumor volume and were compared between patients with tumor control and those with tumor progression. In patients with early posttreatment tumor enlargement, ADC values were compared between patients with pseudoprogression and those with true progression. Of the 44 study patients, 34 (77.3%) demonstrated tumor control at final follow-up. Patients with tumor control had higher pretreatment minimum (1.35 vs 1.09; p = 0.008), mean (1.80 vs 1.45; p = 0.004), and maximum (2.41 vs 1.91; p = 0.011) ADC values than patients with tumor progression. ADC values did not differ between patients with pseudoprogression and those with true progression at early posttreatment follow-up. ADC values may be helpful in predicting response to GKRS in patients with solid VSs but cannot predict which tumors will undergo pseudoprogression. Patients with higher pretreatment ADC values may be more likely to demonstrate posttreatment tumor control.

Sections du résumé

BACKGROUND
Radiosurgery is a well-established treatment for vestibular schwannomas (VSs), but it is often difficult to identify which tumors will respond to treatment. We sought to determine whether pretreatment or posttreatment tumor apparent diffusion coefficient (ADC) values could predict tumor control in patients undergoing Gamma Knife radiosurgery (GKRS) and whether these values could differentiate between cases of pseudoprogression and cases of true progression in the early posttreatment period.
METHODS
We retrospectively identified patients who underwent GKRS for solid VSs between June 2008 and November 2016 and who had a minimum follow-up of 36 months. Pretreatment and posttreatment minimum, mean, and maximum ADC values were measured for the whole tumor volume and were compared between patients with tumor control and those with tumor progression. In patients with early posttreatment tumor enlargement, ADC values were compared between patients with pseudoprogression and those with true progression.
RESULTS
Of the 44 study patients, 34 (77.3%) demonstrated tumor control at final follow-up. Patients with tumor control had higher pretreatment minimum (1.35 vs 1.09; p = 0.008), mean (1.80 vs 1.45; p = 0.004), and maximum (2.41 vs 1.91; p = 0.011) ADC values than patients with tumor progression. ADC values did not differ between patients with pseudoprogression and those with true progression at early posttreatment follow-up.
CONCLUSIONS
ADC values may be helpful in predicting response to GKRS in patients with solid VSs but cannot predict which tumors will undergo pseudoprogression. Patients with higher pretreatment ADC values may be more likely to demonstrate posttreatment tumor control.

Identifiants

pubmed: 33532869
doi: 10.1007/s00701-021-04738-x
pii: 10.1007/s00701-021-04738-x
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1013-1019

Références

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Auteurs

Pranay Soni (P)

Department of Neurological Surgery, Neurological Institute, Cleveland Clinic, Cleveland, OH, USA.
Rose Ella Burkhardt Brain Tumor & Neuro-Oncology Center, Cleveland Clinic, Cleveland, OH, USA.

Tamia Potter (T)

Case Western Reserve University School of Medicine, Cleveland, OH, USA.

Matthew Poturalski (M)

Case Western Reserve University School of Medicine, Cleveland, OH, USA.
Department of Neuroradiology, Cleveland Clinic, Cleveland, OH, USA.

Christopher Karakasis (C)

Case Western Reserve University School of Medicine, Cleveland, OH, USA.
Department of Neuroradiology, Cleveland Clinic, Cleveland, OH, USA.

Hamid Borghei-Razavi (H)

Department of Neurological Surgery, Cleveland Clinic Florida, Weston, FL, USA.

Pablo F Recinos (PF)

Department of Neurological Surgery, Neurological Institute, Cleveland Clinic, Cleveland, OH, USA.
Rose Ella Burkhardt Brain Tumor & Neuro-Oncology Center, Cleveland Clinic, Cleveland, OH, USA.
Case Western Reserve University School of Medicine, Cleveland, OH, USA.

Varun R Kshettry (VR)

Department of Neurological Surgery, Neurological Institute, Cleveland Clinic, Cleveland, OH, USA. kshettv@ccf.org.
Rose Ella Burkhardt Brain Tumor & Neuro-Oncology Center, Cleveland Clinic, Cleveland, OH, USA. kshettv@ccf.org.
Case Western Reserve University School of Medicine, Cleveland, OH, USA. kshettv@ccf.org.

Jonathan Lee (J)

Case Western Reserve University School of Medicine, Cleveland, OH, USA.
Department of Neuroradiology, Cleveland Clinic, Cleveland, OH, USA.

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