Relationship between allergic sensitisation-associated single-nucleotide polymorphisms and allergic transfusion reactions and febrile non-haemolytic transfusion reactions in paediatric cases.


Journal

Blood transfusion = Trasfusione del sangue
ISSN: 2385-2070
Titre abrégé: Blood Transfus
Pays: Italy
ID NLM: 101237479

Informations de publication

Date de publication:
03 2022
Historique:
received: 11 07 2020
accepted: 02 11 2020
pubmed: 5 2 2021
medline: 19 4 2022
entrez: 4 2 2021
Statut: ppublish

Résumé

Allergic transfusion reactions (ATR) and febrile non-haemolytic transfusion reactions (FNHTR) are common transfusion-related adverse reactions; however, their pathogenesis remains unclear and it is difficult to predict their occurrence. Single-nucleotide polymorphisms (SNP) are related to the onset of various diseases and therapy-related adverse events; therefore, identification of SNP related to transfusion-related adverse reactions may help to elucidate the underlying mechanism and predict the onset of these reactions. We retrospectively analysed the association between the onset of ATR or FNHTR and 22 allergic sensitisation-related SNP in 219 children (aged ≤20 years) who had haematological and oncological diseases and who had received transfusions of platelets and/or red blood cell concentrates. Among the 219 children, 105 had developed an ATR and/or FNHTR at least once. The patients who developed ATR frequently had a risk allele in rs6473223, while the patients who developed FNHTR frequently had a risk allele in rs10893845. Furthermore, patients who developed ATR accompanied by febrile symptoms also frequently had a risk allele in rs10893845, similar to patients who developed FNHTR. The results suggested that allergic sensitisation is associated with the onset of ATR and/or FNHTR in some patients. Although further prospective evaluation is necessary, analysis of these SNP might help to provide safer transfusion therapy by predicting patients at higher risk of transfusion-related adverse reactions and further clarifying the pathogenic mechanism underlying such reactions.

Sections du résumé

BACKGROUND
Allergic transfusion reactions (ATR) and febrile non-haemolytic transfusion reactions (FNHTR) are common transfusion-related adverse reactions; however, their pathogenesis remains unclear and it is difficult to predict their occurrence. Single-nucleotide polymorphisms (SNP) are related to the onset of various diseases and therapy-related adverse events; therefore, identification of SNP related to transfusion-related adverse reactions may help to elucidate the underlying mechanism and predict the onset of these reactions.
MATERIALS AND METHODS
We retrospectively analysed the association between the onset of ATR or FNHTR and 22 allergic sensitisation-related SNP in 219 children (aged ≤20 years) who had haematological and oncological diseases and who had received transfusions of platelets and/or red blood cell concentrates.
RESULTS
Among the 219 children, 105 had developed an ATR and/or FNHTR at least once. The patients who developed ATR frequently had a risk allele in rs6473223, while the patients who developed FNHTR frequently had a risk allele in rs10893845. Furthermore, patients who developed ATR accompanied by febrile symptoms also frequently had a risk allele in rs10893845, similar to patients who developed FNHTR.
DISCUSSION
The results suggested that allergic sensitisation is associated with the onset of ATR and/or FNHTR in some patients. Although further prospective evaluation is necessary, analysis of these SNP might help to provide safer transfusion therapy by predicting patients at higher risk of transfusion-related adverse reactions and further clarifying the pathogenic mechanism underlying such reactions.

Identifiants

pubmed: 33539286
pii: 2021.0230-20
doi: 10.2450/2021.0230-20
pmc: PMC8971021
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

94-102

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Auteurs

Yuichiro Ide (Y)

Department of Laboratory Medicine, Shinshu University Hospital, Matsumoto, Japan.
Life Science Research Centre, Nagano Children's Hospital, Azumino, Japan.

Ryu Yanagisawa (R)

Life Science Research Centre, Nagano Children's Hospital, Azumino, Japan.
Division of Blood Transfusion, Shinshu University Hospital, Matsumoto, Japan.
Centre for Advanced Cell Therapy, Shinshu University Hospital, Matsumoto, Japan.

Jun Kobayashi (J)

Department of Laboratory Medicine, Nagano Children's Hospital, Azumino, Japan.

Kazutoshi Komori (K)

Department of Haematology and Oncology, Nagano Children's Hospital, Azumino, Japan.

Kazuyuki Matsuda (K)

Department of Clinical Laboratory Sciences, School of Health Sciences, Shinshu University, Matsumoto, Japan.

Yuji Amano (Y)

Department of Microbiology and Immunology, Shinshu University School of Medicine, Matsumoto, Japan.

Yozo Nakazawa (Y)

Department of Paediatrics, Shinshu University School of Medicine, Matsumoto, Japan.

Toshikazu Takeshita (T)

Department of Microbiology and Immunology, Shinshu University School of Medicine, Matsumoto, Japan.

Kazuo Sakashita (K)

Department of Haematology and Oncology, Nagano Children's Hospital, Azumino, Japan.

Minoru Tozuka (M)

Life Science Research Centre, Nagano Children's Hospital, Azumino, Japan.
Department of Laboratory Medicine, Nagano Children's Hospital, Azumino, Japan.

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