Genetic Creutzfeldt-Jakob disease-M232R with the cooccurrence of multiple prion strains, M1 + M2C + M2T: Report of an autopsy case.


Journal

Neuropathology : official journal of the Japanese Society of Neuropathology
ISSN: 1440-1789
Titre abrégé: Neuropathology
Pays: Australia
ID NLM: 9606526

Informations de publication

Date de publication:
Jun 2021
Historique:
revised: 20 08 2020
received: 24 06 2020
accepted: 07 09 2020
pubmed: 16 2 2021
medline: 26 11 2021
entrez: 15 2 2021
Statut: ppublish

Résumé

Genetic Creutzfeldt-Jakob disease (gCJD) with a methionine to arginine substitution at codon 232 of the prion protein gene (gCJD-M232R) is rare and has only been reported in Japan. We report an autopsy case of gCJD-M232R showing alleles of codon 129 that were homozygous for methionine and the presence of multiple strains of the protease-resistant, abnormal isoform of prion protein (PrP

Identifiants

pubmed: 33586250
doi: 10.1111/neup.12722
doi:

Substances chimiques

PrPSc Proteins 0
Methionine AE28F7PNPL

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

206-213

Informations de copyright

© 2021 Japanese Society of Neuropathology.

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Auteurs

Masayuki Shintaku (M)

Department of Pathology, Hikone Municipal Hospital, Hikone, Japan.

Takeshi Nakamura (T)

Department of Neurology, Kyoto Takeda Hospital, Kyoto, Japan.

Daita Kaneda (D)

Institute of Neuropathology, Fukushimura Hospital, Toyohashi, Japan.

Akiyo Shinde (A)

Department of Neurology, Tenri Hospital, Tenri, Japan.

Hirofumi Kusaka (H)

Department of Neurology, Tanabe Neurosurgical Hospital, Fujiidera, Japan.

Atsuko Takeuchi (A)

Department of Neurological Science, Tohoku University Graduate School of Medicine, Sendai, Japan.

Tetsuyuki Kitamoto (T)

Department of Neurological Science, Tohoku University Graduate School of Medicine, Sendai, Japan.

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