Cardiac conduction abnormalities in patients with degenerated bioprostheses undergoing transcatheter aortic valve-in-valve implantations and their impact on long-term outcomes.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
01 05 2021
Historique:
received: 14 12 2020
revised: 08 02 2021
accepted: 10 02 2021
pubmed: 17 2 2021
medline: 29 5 2021
entrez: 16 2 2021
Statut: ppublish

Résumé

The relationship between preoperative cardiac conduction abnormalities (CCA) and long-term outcomes after transcatheter aortic valve-in-valve implantation (TAVI-VIV) remains unclear. The aim of the study was to evaluate the effects of preoperative CCA on mortality and morbidity after TAVI-VIV and to estimate the impact of new-onset CCA on postoperative outcomes. Between 2011 and 2020, 201 patients with degenerated aortic bioprostheses were qualified for TAVI-VIV procedures in two German heart centers. Cases with previously implanted permanent rhythm-controlling devices were excluded (n = 53). A total of 148 subjects met the eligibility criteria and were divided into 2 study groups according to the presence of preexisting CCA (CCA (n = 84) and non-CCA (n = 64), respectively). Early and late mortality and morbidity were evaluated. Follow-up functional status was assessed according to New York Heart Association (NYHA) classification. There were no procedural deaths. TAVI-VIV related new-onset CCAs were observed in 35.8% patients. The 30-day permanent pacemaker implantation rate was 1.6% in non-CCA vs 9.5% in CCA group (p = 0.045). Preexisting right bundle-branch block (OR:5.01; 95%CI, 1.05-23.84) and first-degree atrioventricular block (OR:4.55; 95%CI, 1.10-18.73) were independent predictors of new pacemaker implantation. One-year and five-year probability of survival were comparable in CCA and non-CCA groups: 90.3% vs 91.8% and 68.2% vs 74.3%, respectively. Surviving patients with preexisting and new-onset CCA had a worse functional status according to NYHA classification at follow-up. Preexisting and new-onset postoperative CCAs did not affect early and late mortality after TAVI-VIV procedures, however, they may have a negative impact on late functional status.

Sections du résumé

BACKGROUND
The relationship between preoperative cardiac conduction abnormalities (CCA) and long-term outcomes after transcatheter aortic valve-in-valve implantation (TAVI-VIV) remains unclear. The aim of the study was to evaluate the effects of preoperative CCA on mortality and morbidity after TAVI-VIV and to estimate the impact of new-onset CCA on postoperative outcomes.
METHODS
Between 2011 and 2020, 201 patients with degenerated aortic bioprostheses were qualified for TAVI-VIV procedures in two German heart centers. Cases with previously implanted permanent rhythm-controlling devices were excluded (n = 53). A total of 148 subjects met the eligibility criteria and were divided into 2 study groups according to the presence of preexisting CCA (CCA (n = 84) and non-CCA (n = 64), respectively). Early and late mortality and morbidity were evaluated. Follow-up functional status was assessed according to New York Heart Association (NYHA) classification.
RESULTS
There were no procedural deaths. TAVI-VIV related new-onset CCAs were observed in 35.8% patients. The 30-day permanent pacemaker implantation rate was 1.6% in non-CCA vs 9.5% in CCA group (p = 0.045). Preexisting right bundle-branch block (OR:5.01; 95%CI, 1.05-23.84) and first-degree atrioventricular block (OR:4.55; 95%CI, 1.10-18.73) were independent predictors of new pacemaker implantation. One-year and five-year probability of survival were comparable in CCA and non-CCA groups: 90.3% vs 91.8% and 68.2% vs 74.3%, respectively. Surviving patients with preexisting and new-onset CCA had a worse functional status according to NYHA classification at follow-up.
CONCLUSION
Preexisting and new-onset postoperative CCAs did not affect early and late mortality after TAVI-VIV procedures, however, they may have a negative impact on late functional status.

Identifiants

pubmed: 33592238
pii: S0167-5273(21)00282-5
doi: 10.1016/j.ijcard.2021.02.029
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

16-22

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2021 Elsevier B.V. All rights reserved.

Auteurs

Tomasz Stankowski (T)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany. Electronic address: tomekstankowski89@gmail.com.

Norman Mangner (N)

Herzzentrum Dresden, Technische Universität Dresden, Department of Internal Medicine and Cardiology, Dresden, Germany.

Axel Linke (A)

Herzzentrum Dresden, Technische Universität Dresden, Department of Internal Medicine and Cardiology, Dresden, Germany.

Sleiman Sebastian Aboul-Hassan (SS)

Department of Cardiac Surgery, Medinet Heart Center Ltd., Nowa Sol, Poland.

Tomasz Gąsior (T)

Herzzentrum Dresden, Technische Universität Dresden, Department of Internal Medicine and Cardiology, Dresden, Germany.

Anja Muehle (A)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Volker Herwig (V)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Axel Harnath (A)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Mohammed Salem (M)

Department of Cardiology, Carl-Thiem-Klinikum, Cottbus, Germany.

Michał Szłapka (M)

Department of Cardiac Surgery, Asklepios Klinik Harburg, Hamburg, Germany.

Oliver Grimmig (O)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Soeren Just (S)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Dirk Fritzsche (D)

Sana Heart Center Cottbus, Department of Cardiac Surgery, Cottbus, Germany.

Bartłomiej Perek (B)

Department of Cardiac Surgery and Transplantology, Poznan University of Medical Sciences, Poznan, Poland.

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