Loss of CASK Accelerates Heart Failure Development.

Animals Calcium / metabolism Calcium-Calmodulin-Dependent Protein Kinase Type 2 / metabolism Cells, Cultured Glucagon-Like Peptide-1 Receptor / metabolism Guanylate Kinases / genetics Heart Failure / genetics Heart Rate Humans Male Mice Mice, Inbred C57BL Myocardial Contraction Myocytes, Cardiac / metabolism
calmodulin heart failure myocardium neurons sarcoplasmic reticulum

Journal

Circulation research
ISSN: 1524-4571
Titre abrégé: Circ Res
Pays: United States
ID NLM: 0047103

Informations de publication

Date de publication:
16 04 2021
Historique:
pubmed: 18 2 2021
medline: 8 1 2022
entrez: 17 2 2021
Statut: ppublish

Résumé

[Figure: see text].

Identifiants

DOI: 10.1161/CIRCRESAHA.120.318170 PMID: 33593074 PMC: PMC8049978
pubmed: 33593074
doi: 10.1161/CIRCRESAHA.120.318170
pmc: PMC8049978
mid: NIHMS1675222
doi:

Substances chimiques

Glucagon-Like Peptide-1 Receptor 0
CASK kinases EC 2.7.11.1
Calcium-Calmodulin-Dependent Protein Kinase Type 2 EC 2.7.11.17
Guanylate Kinases EC 2.7.4.8
Calcium SY7Q814VUP

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1139-1155

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL079031
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL096652
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL092097
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL113001
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL070250
Pays : United States
Organisme : NHLBI NIH HHS
ID : R35 HL140034
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL142282
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL134824
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL114383
Pays : United States
Organisme : NHLBI NIH HHS
ID : R35 HL135754
Pays : United States

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Auteurs

Julian Mustroph (J)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Can M Sag (CM)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Felix Bähr (F)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Anna-Lena Schmidtmann (AL)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Shamindra N Gupta (SN)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Alexander Dietz (A)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

M M Towhidul Islam (MMT)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Charlotte Lücht (C)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Bo Eric Beuthner (BE)

Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Steffen Pabel (S)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Maria J Baier (MJ)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Ali El-Armouche (A)

Department of Pharmacology and Toxicology, Technical University Dresden, Germany (A.E.-A.).

Samuel Sossalla (S)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).
Cardiology & Pneumology, University Medical Center Göttingen, Germany (F.B., A.-L.S., S.N.G., A.D., M.M.T.I., B.E.B., S.S.).

Mark E Anderson (ME)

Johns Hopkins School of Medicine, Baltimore (M.E.A.).

Julia Möllmann (J)

Clinic for Cardiology, Angiology, and Internal Intensive Care, University Clinic Aachen, Germany (J. Möllmann, M.L., N.M.).

Michael Lehrke (M)

Clinic for Cardiology, Angiology, and Internal Intensive Care, University Clinic Aachen, Germany (J. Möllmann, M.L., N.M.).

Nikolaus Marx (N)

Clinic for Cardiology, Angiology, and Internal Intensive Care, University Clinic Aachen, Germany (J. Möllmann, M.L., N.M.).

Peter J Mohler (PJ)

College of Medicine, the Ohio State University Wexner Medical Center, Columbus (P.J.M.).

Donald M Bers (DM)

College of Biological Sciences, University of California at Davis (D.M.B.).

Bernhard Unsöld (B)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Tao He (T)

Department of Molecular Cardiology and Epigenetics, University Clinic Heidelberg, Germany (T.H., M.D., J.B.).

Matthias Dewenter (M)

Department of Molecular Cardiology and Epigenetics, University Clinic Heidelberg, Germany (T.H., M.D., J.B.).

Johannes Backs (J)

Department of Molecular Cardiology and Epigenetics, University Clinic Heidelberg, Germany (T.H., M.D., J.B.).

Lars S Maier (LS)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

Stefan Wagner (S)

Department of Internal Medicine II, University Medical Center Regensburg, Germany (J. Mustroph, C.M.S., C.L., S.P., M.J.B., S.S., B.U., L.S.M., S.W.).

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Classifications MeSH

questionsmedicales.fr › Eucaryotes › Animaux › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Facteurs biologiques › Facteurs de la coagulation sanguine › Calcium › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Acides aminés, peptides et protéines › Protéines › Protéines et peptides de signalisation intracellulaire › Protein-Serine-Threonine Kinases › Calcium-Calmodulin-Dependent Protein Kinases › Calcium-Calmodulin-Dependent Protein Kinase Type 2 › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Cellules › Cellules cultivées › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Acides aminés, peptides et protéines › Protéines › Protéines membranaires › Récepteurs de surface cellulaire › Récepteurs peptidiques › Récepteur hormone gastrointestinale › Récepteurs des peptides similaires au glucagon › Récepteur du peptide-1 similaire au glucagon › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Enzymes et coenzymes › Enzymes › Transferases › Phosphotransferases › Phosphotransferases (Phosphate Group Acceptor) › Guanylate kinase › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Maladies cardiovasculaires › Cardiopathies › Défaillance cardiaque › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Phénomènes physiologiques respiratoires et circulatoires › Phénomènes physiologiques cardiovasculaires › Hémodynamique › Rythme cardiaque › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Eucaryotes › Animaux › Chordés › Vertébrés › Mammifères › Eutheria › Primates › Haplorhini › Catarrhini › Hominidae › Humains › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Eucaryotes › Animaux › Chordés › Vertébrés › Mammifères › Eutheria › Rodentia › Muridae › Murinae › Souris › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Eucaryotes › Animaux › Chordés › Vertébrés › Mammifères › Eutheria › Rodentia › Muridae › Murinae › Souris › Lignées consanguines de souris › Souris de lignée C57BL › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Phénomènes physiologiques de l'appareil locomoteur et du système nerveux › Phénomènes physiologiques du système locomoteur › Contraction musculaire › Contraction myocardique › Loss of CASK Accelerates Heart Failure Development.
questionsmedicales.fr › Cellules › Cellules musculaires › Myocytes cardiaques › Loss of CASK Accelerates Heart Failure Development.