Prognostic Value of Early Risk Stratification in Pediatric Pulmonary Arterial Hypertension.


Journal

Transplantation proceedings
ISSN: 1873-2623
Titre abrégé: Transplant Proc
Pays: United States
ID NLM: 0243532

Informations de publication

Date de publication:
Jun 2021
Historique:
pubmed: 25 2 2021
medline: 14 7 2021
entrez: 24 2 2021
Statut: ppublish

Résumé

Pulmonary arterial hypertension (PAH) is a life-threatening disease with risk stratification-based treatment strategy in adults. Although the risk factors have been studied individually in children, effective risk stratification is still lacking. We have tested the prognostic accuracy of pediatric PAH risk factors in our patient group. Records of 58 PAH patients treated between 1995 and 2019 were reviewed retrospectively. Median age at diagnosis was 4.2 years (range, 0.1-16.1 years), and follow-up was 5.4 years (range, 0.01-24.1 years). Data collected at diagnosis were demographics, World Health Organization functional class, evidence of right ventricular failure, and parameters of echocardiography and cardiac catheterization. Mortality was 29% and 33% reached the composite endpoint. Patients with idiopathic PAH (n = 12) had increased risk of mortality compared with the congenital heart disease-associated PAH group (n = 32) (P = .0024). Neither the initial World Health Organization functional class staging nor the echocardiographic parameters significantly predicted the prognosis. The number of risk factors had no significant prognostic value either. In contrast, patients with higher pulmonary vascular resistance index (PVRI) had significantly increased risk (each 10 Wood units ⋅ m Survival analysis showed that PAH etiology might be an important determinant in pediatric PAH risk stratification. We confirmed that PVRI has predictive value in prognostic assessment. We could not establish the prognostic value of nonweighted single risk factors or their combination to predict pediatric PAH outcome due to the low sample size, but these results indicate that such studies are warranted.

Sections du résumé

BACKGROUND BACKGROUND
Pulmonary arterial hypertension (PAH) is a life-threatening disease with risk stratification-based treatment strategy in adults. Although the risk factors have been studied individually in children, effective risk stratification is still lacking. We have tested the prognostic accuracy of pediatric PAH risk factors in our patient group.
PATIENTS AND METHODS METHODS
Records of 58 PAH patients treated between 1995 and 2019 were reviewed retrospectively. Median age at diagnosis was 4.2 years (range, 0.1-16.1 years), and follow-up was 5.4 years (range, 0.01-24.1 years). Data collected at diagnosis were demographics, World Health Organization functional class, evidence of right ventricular failure, and parameters of echocardiography and cardiac catheterization.
RESULTS RESULTS
Mortality was 29% and 33% reached the composite endpoint. Patients with idiopathic PAH (n = 12) had increased risk of mortality compared with the congenital heart disease-associated PAH group (n = 32) (P = .0024). Neither the initial World Health Organization functional class staging nor the echocardiographic parameters significantly predicted the prognosis. The number of risk factors had no significant prognostic value either. In contrast, patients with higher pulmonary vascular resistance index (PVRI) had significantly increased risk (each 10 Wood units ⋅ m
CONCLUSIONS CONCLUSIONS
Survival analysis showed that PAH etiology might be an important determinant in pediatric PAH risk stratification. We confirmed that PVRI has predictive value in prognostic assessment. We could not establish the prognostic value of nonweighted single risk factors or their combination to predict pediatric PAH outcome due to the low sample size, but these results indicate that such studies are warranted.

Identifiants

pubmed: 33622612
pii: S0041-1345(21)00081-6
doi: 10.1016/j.transproceed.2021.01.047
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1439-1442

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

László Ablonczy (L)

Gottsegen György National Institute of Cardiology, Pediatric Heart Center, Budapest, Hungary. Electronic address: ablonczyl@gmail.com.

Tamás Ferenci (T)

Óbuda University, Physiological Controls Research Center, Budapest, Hungary.

Orsolya Somoskövi (O)

Gottsegen György National Institute of Cardiology, Pediatric Heart Center, Budapest, Hungary.

Réka Osváth (R)

Gottsegen György National Institute of Cardiology, Pediatric Heart Center, Budapest, Hungary.

György S Reusz (GS)

1st Department of Pediatrics, Semmelweis University, Budapest, Hungary.

Eva Kis (E)

Gottsegen György National Institute of Cardiology, Pediatric Heart Center, Budapest, Hungary.

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