Dynamic left ventricular outflow tract obstruction in a patient with acute coronary syndrome and without the apical akinesia: Potential alternative mechanisms causing a dynamic left ventricular outflow tract obstruction other than a compensatory basal hyperkinesis.

acute coronary syndrome apical akinesia compensatory basal hyperkinesis left ventricular outflow tract obstruction

Journal

Echocardiography (Mount Kisco, N.Y.)
ISSN: 1540-8175
Titre abrégé: Echocardiography
Pays: United States
ID NLM: 8511187

Informations de publication

Date de publication:
03 2021
Historique:
revised: 09 11 2020
received: 24 08 2020
accepted: 11 01 2021
pubmed: 26 2 2021
medline: 10 7 2021
entrez: 25 2 2021
Statut: ppublish

Résumé

The mechanism for dynamic left ventricular outflow tract obstruction (LVOTO) after acute coronary syndromes (ACS) is thought to be apical infarction with compensatory hyperkinesia of the residual normally perfused basal segments of the myocardium. However, herein, we report a patient with ACS and dynamic LVOTO (peak gradient of 250 mm Hg at rest) that could not be secondary to apical akinesia. We propose a potential alternative mechanism leading to dynamic LVOTO in ACS, namely, the interplay between sigmoid septum, basal hyperkinesis, and outflow tract narrowing induced by afterload reduction due to acute myocardial ischemia itself.

Identifiants

pubmed: 33629388
doi: 10.1111/echo.14989
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

460-468

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

Dimitrow PP, Bober M, Michałowska J, et al Left ventricular outflow tract gradient provoked by upright position or exercise in treated patients with hypertrophic cardiomyopathy without obstruction at rest. Echocardiography. 2009;26(5):513-520.
Kawai K, Sengoku H, Ishihara H, et al Total clinical course and autopsy findings of left ventricular outflow tract obstruction due to sigmoid septum: histologically proven isolated basal septal hypertrophy. Intern Med. 2017;56(16):2151-2154.
Armstrong WF, Marcovitz PA. Dynamic left ventricular outflow tract obstruction as a complication of acute myocardial infarction. Am Heart J. 1996;131(4):827-830.
Haley JH, Sinak LJ, Tajik AJ, et al Dynamic left ventricular outflow tract obstruction in acute coronary syndromes: an important cause of new systolic murmur and cardiogenic shock. Mayo Clin Proc. 1999;74(9):901-906.
Joffe II, Riley MF, Katz SE, et al Acquired dynamic left ventricular outflow tract obstruction complicating acute anterior myocardial infarction: serial echocardiographic and clinical evaluation. J Am Soc Echocardiogr. 1997;10(7):717-721.
Meuwese CL, Boulaksil M, van Dijk J, et al Transient left ventricular outflow tract obstruction with systolic anterior motion of the mitral valve: A stunning cause. Echocardiography. 2017;34(7):1089-1091.
Ozaki K, Okubo T, Yano T, et al Manifestation of latent left ventricular outflow tract obstruction caused by acute myocardial infarction: An important complication of acute myocardial infarction. J Cardiol. 2015;65(6):514-518.
Takamura T, Dohi K, Satomi A, et al Intra-aortic balloon pump induced dynamic left ventricular outflow tract obstruction and cardiogenic shock after very late stent thrombosis in the left anterior descending coronary artery. J Cardiol Cases. 2012;6(5):e137-e140.
Grines CL, Topol EJ, Califf RM, et al Prognostic implications and predictors of enhanced regional wall motion of the noninfarct zone after thrombolysis and angioplasty therapy of acute myocardial infarction. The TAMI Study Groups. Circulation. 1989;80(2):245-253.
Buda AJ, Lefkowitz CA, Gallagher KP. Augmentation of regional function in nonischemic myocardium during coronary occlusion measured with two-dimensional echocardiography. J Am Coll Cardiol. 1990;16(1):175-180.
Goto Y, Igarashi Y, Yamada O, et al Hyperkinesis without the Frank-Starling mechanism in a nonischemic region of acutely ischemic excised canine heart. Circulation. 1988;77(2):468-477.

Auteurs

Momo Fujiwara (M)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Keisuke Kawai (K)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Natsuki Kanazawa (N)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Masamitsu Noguchi (M)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Minoru Hasokawa (M)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Masakazu Nanahoshi (M)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

Seiichi Kobayashi (S)

Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.

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