Targeting RAGE to prevent SARS-CoV-2-mediated multiple organ failure: Hypotheses and perspectives.
Animals
COVID-19
/ complications
Drug Discovery
Humans
Molecular Targeted Therapy
Multiple Organ Failure
/ etiology
Receptor for Advanced Glycation End Products
/ antagonists & inhibitors
Renin-Angiotensin System
/ drug effects
SARS-CoV-2
/ drug effects
Signal Transduction
/ drug effects
COVID-19 Drug Treatment
COVID-19
HMGB1
RAGE
Renin-angiotensin system (RAS)
SARS-CoV-2
Journal
Life sciences
ISSN: 1879-0631
Titre abrégé: Life Sci
Pays: Netherlands
ID NLM: 0375521
Informations de publication
Date de publication:
01 May 2021
01 May 2021
Historique:
received:
04
09
2020
revised:
12
02
2021
accepted:
18
02
2021
pubmed:
27
2
2021
medline:
25
3
2021
entrez:
26
2
2021
Statut:
ppublish
Résumé
A novel infectious disease (COVID-19), caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), was detected in December 2019 and declared as a global pandemic by the World Health. Approximately 15% of patients with COVID-19 progress to severe pneumonia and eventually develop acute respiratory distress syndrome (ARDS), septic shock and/or multiple organ failure with high morbidity and mortality. Evidence points towards a determinant pathogenic role of members of the renin-angiotensin system (RAS) in mediating the susceptibility, infection, inflammatory response and parenchymal injury in lungs and other organs of COVID-19 patients. The receptor for advanced glycation end-products (RAGE), a member of the immunoglobulin superfamily, has important roles in pulmonary pathological states, including fibrosis, pneumonia and ARDS. RAGE overexpression/hyperactivation is essential to the deleterious effects of RAS in several pathological processes, including hypertension, chronic kidney and cardiovascular diseases, and diabetes, all of which are major comorbidities of SARS-CoV-2 infection. We propose RAGE as an additional molecular target in COVID-19 patients for ameliorating the multi-organ pathology induced by the virus and improving survival, also in the perspective of future infections by other coronaviruses.
Identifiants
pubmed: 33636175
pii: S0024-3205(21)00236-8
doi: 10.1016/j.lfs.2021.119251
pmc: PMC7900755
pii:
doi:
Substances chimiques
Receptor for Advanced Glycation End Products
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
119251Informations de copyright
Copyright © 2021 Elsevier Inc. All rights reserved.
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