Pancreatoduodenectomy for Neuroendocrine Tumors in Patients with Multiple Endocrine Neoplasia Type 1: An AFCE (Association Francophone de Chirurgie Endocrinienne) and GTE (Groupe d'étude des Tumeurs Endocrines) Study.


Journal

World journal of surgery
ISSN: 1432-2323
Titre abrégé: World J Surg
Pays: United States
ID NLM: 7704052

Informations de publication

Date de publication:
06 2021
Historique:
accepted: 30 01 2021
pubmed: 3 3 2021
medline: 9 7 2021
entrez: 2 3 2021
Statut: ppublish

Résumé

To assess postoperative complications and control of hormone secretions following pancreatoduodenectomy (PD) performed on multiple endocrine neoplasia type 1 (MEN1) patients with duodenopancreatic neuroendocrine tumors (DP-NETs). The use of PD to treat MEN1 remains controversial, and evaluating the right place of PD in MEN1 disease makes sense. Thirty-one MEN1 patients from the Groupe d'étude des Tumeurs Endocrines MEN1 cohort who underwent PD for DP-NETs between 1971 and 2013 were included. Early and late postoperative complications, secretory control and overall survival were analyzed. Indication for surgery was: Zollinger-Ellison syndrome (n = 18; 58%), nonfunctioning tumor (n = 9; 29%), insulinoma (n = 2; 7%), VIPoma (n = 1; 3%) and glucagonoma (n = 1; 3%). Mean follow-up was 141 months (range 0-433). Pancreatic fistulas occurred in 5 patients (16.1%), distant metastases in 6 (mean onset of 43 months; range 13-110 months), postoperative diabetes mellitus in 7 (22%), and pancreatic exocrine insufficiency in 6 (19%). Five-year overall survival was 93.3% [CI 75.8-98.3] and ten-year overall survival was 89.1% [CI 69.6-96.4]. After a mean follow-up of 151 months (range 0-433), the biochemical cure rate for MEN-1 related gastrinomas was 61%. In MEN1 patients, pancreatoduodenectomy can be used to control hormone secretions (gastrin, glucagon, VIP) and to remove large NETs. PD was found to control gastrin secretions in about 60% of cases.

Sections du résumé

AIM
To assess postoperative complications and control of hormone secretions following pancreatoduodenectomy (PD) performed on multiple endocrine neoplasia type 1 (MEN1) patients with duodenopancreatic neuroendocrine tumors (DP-NETs).
BACKGROUND
The use of PD to treat MEN1 remains controversial, and evaluating the right place of PD in MEN1 disease makes sense.
METHODS
Thirty-one MEN1 patients from the Groupe d'étude des Tumeurs Endocrines MEN1 cohort who underwent PD for DP-NETs between 1971 and 2013 were included. Early and late postoperative complications, secretory control and overall survival were analyzed.
RESULTS
Indication for surgery was: Zollinger-Ellison syndrome (n = 18; 58%), nonfunctioning tumor (n = 9; 29%), insulinoma (n = 2; 7%), VIPoma (n = 1; 3%) and glucagonoma (n = 1; 3%). Mean follow-up was 141 months (range 0-433). Pancreatic fistulas occurred in 5 patients (16.1%), distant metastases in 6 (mean onset of 43 months; range 13-110 months), postoperative diabetes mellitus in 7 (22%), and pancreatic exocrine insufficiency in 6 (19%). Five-year overall survival was 93.3% [CI 75.8-98.3] and ten-year overall survival was 89.1% [CI 69.6-96.4]. After a mean follow-up of 151 months (range 0-433), the biochemical cure rate for MEN-1 related gastrinomas was 61%.
CONCLUSION
In MEN1 patients, pancreatoduodenectomy can be used to control hormone secretions (gastrin, glucagon, VIP) and to remove large NETs. PD was found to control gastrin secretions in about 60% of cases.

Identifiants

pubmed: 33649917
doi: 10.1007/s00268-021-06005-7
pii: 10.1007/s00268-021-06005-7
pmc: PMC8093175
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1794-1802

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Auteurs

Nicolas Santucci (N)

Department of Digestive and Endocrine Surgery, Dijon University Hospital, University of Burgundy, Dijon, France. nicolas.santucci@chu-dijon.fr.
INSERM, U1231, EPICAD Team UMR "Lipids, Nutrition, Cancer", Dijon, France. nicolas.santucci@chu-dijon.fr.
Service de Chirurgie Digestive, Cancérologique et Endocrinienne, CHU « François Mitterrand », 14, rue Paul Gaffarel, 21079, Dijon Cedex, France. nicolas.santucci@chu-dijon.fr.

Sébastien Gaujoux (S)

Department of Pancreatic and Endocrine Surgery, Cochin University Hospital, APHP, Paris, France.

Christine Binquet (C)

INSERM, U1231, EPICAD Team UMR "Lipids, Nutrition, Cancer", Dijon, France.
INSERM, CIC1432, Clinical Epidemiology Unit, University of Burgundy-Franche-Comté, Dijon, France.

Cynthia Reichling (C)

Department of Hepatogastroenterology and Digestive Oncology, Dijon University Hospital, University of Burgundy, Dijon Cedex, France.

Jean-Christophe Lifante (JC)

Department of General, Digestive and Endocrine Surgery, University Hospital of Lyon Sud and EA 7425 HESPER, Health Services and Performance Research, University Claude-Bernard Lyon 1, Lyon, France.

Bruno Carnaille (B)

Department of General and Endocrine Surgery, Lille University Hospital, Univ. Lille, INSERM U1190, Lille, France.

François Pattou (F)

Department of General and Endocrine Surgery, Lille University Hospital, Univ. Lille, INSERM U1190, Lille, France.

Eric Mirallié (E)

Department of General and Endocrine Surgery, University Hospital of Nantes, Nantes, France.

Olivier Facy (O)

Department of Digestive and Endocrine Surgery, Dijon University Hospital, University of Burgundy, Dijon, France.

Muriel Mathonnet (M)

Department of General, Digestive and Endocrine Surgery, Dupuytren University Hospital, Limoges, France.

Pierre Goudet (P)

Department of Digestive and Endocrine Surgery, Dijon University Hospital, University of Burgundy, Dijon, France.
INSERM, U1231, EPICAD Team UMR "Lipids, Nutrition, Cancer", Dijon, France.

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