Insulin-stimulated endoproteolytic TUG cleavage links energy expenditure with glucose uptake.


Journal

Nature metabolism
ISSN: 2522-5812
Titre abrégé: Nat Metab
Pays: Germany
ID NLM: 101736592

Informations de publication

Date de publication:
03 2021
Historique:
received: 31 10 2019
accepted: 05 02 2021
pubmed: 10 3 2021
medline: 24 4 2021
entrez: 9 3 2021
Statut: ppublish

Résumé

TUG tethering proteins bind and sequester GLUT4 glucose transporters intracellularly, and insulin stimulates TUG cleavage to translocate GLUT4 to the cell surface and increase glucose uptake. This effect of insulin is independent of phosphatidylinositol 3-kinase, and its physiological relevance remains uncertain. Here we show that this TUG cleavage pathway regulates both insulin-stimulated glucose uptake in muscle and organism-level energy expenditure. Using mice with muscle-specific Tug (Aspscr1)-knockout and muscle-specific constitutive TUG cleavage, we show that, after GLUT4 release, the TUG C-terminal cleavage product enters the nucleus, binds peroxisome proliferator-activated receptor (PPAR)γ and its coactivator PGC-1α and regulates gene expression to promote lipid oxidation and thermogenesis. This pathway acts in muscle and adipose cells to upregulate sarcolipin and uncoupling protein 1 (UCP1), respectively. The PPARγ2 Pro12Ala polymorphism, which reduces diabetes risk, enhances TUG binding. The ATE1 arginyltransferase, which mediates a specific protein degradation pathway and controls thermogenesis, regulates the stability of the TUG product. We conclude that insulin-stimulated TUG cleavage coordinates whole-body energy expenditure with glucose uptake, that this mechanism might contribute to the thermic effect of food and that its attenuation could promote obesity.

Identifiants

pubmed: 33686286
doi: 10.1038/s42255-021-00359-x
pii: 10.1038/s42255-021-00359-x
pmc: PMC7990718
mid: NIHMS1671263
doi:

Substances chimiques

Aspscr1 protein, mouse 0
Insulin 0
Intracellular Signaling Peptides and Proteins 0
PPAR gamma 0
Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha 0
Ppargc1a protein, mouse 0
Aminoacyltransferases EC 2.3.2.-
Ate1 protein, mouse EC 2.3.2.-
Glucose IY9XDZ35W2

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

378-393

Subventions

Organisme : NIDDK NIH HHS
ID : R01 DK092661
Pays : United States
Organisme : NIDDK NIH HHS
ID : R56 DK092661
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK114793
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK116774
Pays : United States
Organisme : NIDDK NIH HHS
ID : P30 DK045735
Pays : United States
Organisme : NHLBI NIH HHS
ID : K99 HL150234
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001863
Pays : United States
Organisme : NIGMS NIH HHS
ID : T32 GM136651
Pays : United States
Organisme : NIDDK NIH HHS
ID : F30 DK115037
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK124272
Pays : United States

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Auteurs

Estifanos N Habtemichael (EN)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Evelo Biosciences, Inc., Cambridge, MA, USA.

Don T Li (DT)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Department of Cell Biology, Yale School of Medicine, New Haven, CT, USA.

João Paulo Camporez (JP)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
University of São Paulo, São Paulo, Brazil.

Xavier O Westergaard (XO)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Columbia University, New York, NY, USA.

Chloe I Sales (CI)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Xinran Liu (X)

Department of Cell Biology, Yale School of Medicine, New Haven, CT, USA.

Francesc López-Giráldez (F)

Yale Center for Genome Analysis, Yale School of Medicine, New Haven, CT, USA.

Stephen G DeVries (SG)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Hanbing Li (H)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Zhejiang University of Technology, Hangzhou, China.

Diana M Ruiz (DM)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Kenny Y Wang (KY)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Bhavesh S Sayal (BS)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Sofia González Zapata (S)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Pamela Dann (P)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Stacey N Brown (SN)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Sandro Hirabara (S)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Institute of Physical Activity Sciences and Sports, Cruzeiro do Sul University, São Paulo, Brazil.

Daniel F Vatner (DF)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Leigh Goedeke (L)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

William Philbrick (W)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.

Gerald I Shulman (GI)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA.
Department of Cellular and Molecular Physiology, Yale School of Medicine, New Haven, CT, USA.

Jonathan S Bogan (JS)

Section of Endocrinology and Metabolism, Department of Internal Medicine, Yale School of Medicine, New Haven, CT, USA. jonathan.bogan@yale.edu.
Department of Cell Biology, Yale School of Medicine, New Haven, CT, USA. jonathan.bogan@yale.edu.

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