Re-appraisal of the obesity paradox in heart failure: a meta-analysis of individual data.


Journal

Clinical research in cardiology : official journal of the German Cardiac Society
ISSN: 1861-0692
Titre abrégé: Clin Res Cardiol
Pays: Germany
ID NLM: 101264123

Informations de publication

Date de publication:
Aug 2021
Historique:
received: 14 12 2020
accepted: 11 02 2021
pubmed: 12 3 2021
medline: 30 12 2021
entrez: 11 3 2021
Statut: ppublish

Résumé

Higher body mass index (BMI) is associated with better outcome compared with normal weight in patients with HF and other chronic diseases. It remains uncertain whether the apparent protective role of obesity relates to the absence of comorbidities. Therefore, we investigated the effect of BMI on outcome in younger patients without co-morbidities as compared to older patients with co-morbidities in a large heart failure (HF) population. In an individual patient data analysis from pooled cohorts, 5,819 patients with chronic HF and data available on BMI, co-morbidities and outcome were analysed. Patients were divided into four groups based on BMI (i.e. ≤ 18.5 kg/m Mean age was 65 ± 12 years, with a majority of males (78%), ischaemic HF and HF with reduced ejection fraction. Frequency of all-cause mortality or HF hospitalization was significantly worse in the lowest two BMI groups as compared to the other two groups; however, this effect was only seen in patients older than 75 years or having at least one relevant co-morbidity, and not in younger patients with HF only. After including medications and N-terminal pro-B-type natriuretic peptide and high-sensitivity cardiac troponin concentrations into the model, the prognostic impact of BMI was largely absent even in the elderly group with co-morbidity. The present study suggests that obesity is a marker of less advanced disease, but does not have an independent protective effect in patients with chronic HF. Categories of BMI are only predictive of poor outcome in patients aged > 75 years or with at least one co-morbidity (bottom), but not in those aged < 75 years without co-morbidities (top). The prognostic effect largely disappears in multivariable analyses even for the former group. These findings question the protective effect of obesity in chronic heart failure (HF).

Sections du résumé

BACKGROUND BACKGROUND
Higher body mass index (BMI) is associated with better outcome compared with normal weight in patients with HF and other chronic diseases. It remains uncertain whether the apparent protective role of obesity relates to the absence of comorbidities. Therefore, we investigated the effect of BMI on outcome in younger patients without co-morbidities as compared to older patients with co-morbidities in a large heart failure (HF) population.
METHODS METHODS
In an individual patient data analysis from pooled cohorts, 5,819 patients with chronic HF and data available on BMI, co-morbidities and outcome were analysed. Patients were divided into four groups based on BMI (i.e. ≤ 18.5 kg/m
RESULTS RESULTS
Mean age was 65 ± 12 years, with a majority of males (78%), ischaemic HF and HF with reduced ejection fraction. Frequency of all-cause mortality or HF hospitalization was significantly worse in the lowest two BMI groups as compared to the other two groups; however, this effect was only seen in patients older than 75 years or having at least one relevant co-morbidity, and not in younger patients with HF only. After including medications and N-terminal pro-B-type natriuretic peptide and high-sensitivity cardiac troponin concentrations into the model, the prognostic impact of BMI was largely absent even in the elderly group with co-morbidity.
CONCLUSIONS CONCLUSIONS
The present study suggests that obesity is a marker of less advanced disease, but does not have an independent protective effect in patients with chronic HF. Categories of BMI are only predictive of poor outcome in patients aged > 75 years or with at least one co-morbidity (bottom), but not in those aged < 75 years without co-morbidities (top). The prognostic effect largely disappears in multivariable analyses even for the former group. These findings question the protective effect of obesity in chronic heart failure (HF).

Identifiants

pubmed: 33704552
doi: 10.1007/s00392-021-01822-1
pii: 10.1007/s00392-021-01822-1
pmc: PMC8318940
doi:

Substances chimiques

Biomarkers 0
Peptide Fragments 0
Troponin 0
pro-brain natriuretic peptide (1-76) 0
Natriuretic Peptide, Brain 114471-18-0

Types de publication

Journal Article Meta-Analysis

Langues

eng

Sous-ensembles de citation

IM

Pagination

1280-1291

Informations de copyright

© 2021. The Author(s).

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Auteurs

Nick Marcks (N)

Department of Cardiology, Maastricht University Medical Centre, PO Box 5800, 6202AZ, Maastricht, The Netherlands.

Alberto Aimo (A)

Cardiology Division, University Hospital of Pisa, Pisa, Italy.

James L Januzzi (JL)

Massachusetts General Hospital and Baim Institute for Clinical Research, Boston, USA.

Giuseppe Vergaro (G)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.
Fondazione Toscana G. Monasterio, Pisa, Italy.

Aldo Clerico (A)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.
Fondazione Toscana G. Monasterio, Pisa, Italy.

Roberto Latini (R)

Department of Cardiovascular Medicine, Institute for Pharmacological Research Mario Negri IRCCS, Milan, Italy.

Jennifer Meessen (J)

Department of Cardiovascular Medicine, Institute for Pharmacological Research Mario Negri IRCCS, Milan, Italy.

Inder S Anand (IS)

Division of Cardiovascular Medicine, University of Minnesota, Minneapolis, USA.
Department of Cardiology, VA Medical Centre, Minneapolis, USA.

Jay N Cohn (JN)

Division of Cardiovascular Medicine, University of Minnesota, Minneapolis, USA.

Jørgen Gravning (J)

Department of Cardiology, Oslo University Hospital, Ullevål, Oslo, Norway.
Centre for Heart Failure Research, University of Oslo, Oslo, Norway.

Thor Ueland (T)

Research Institute of Internal Medicine, Oslo University Hospital, Rikshospitalet, Oslo, Norway.
Faculty of Medicine, University of Oslo, Oslo, Norway.
K. G. Jebsen Thrombosis Research and Expertise Centre, University of Tromsø, Tromsø, Norway.

Antoni Bayes-Genis (A)

Hospital Universitari Germans Trias I Pujol, Badalona (Barcelona), Spain.

Josep Lupón (J)

Hospital Universitari Germans Trias I Pujol, Badalona (Barcelona), Spain.

Rudolf A de Boer (RA)

University Medical Centre Groningen, University Medical Centre Groningen, Groningen, The Netherlands.

Akiomi Yoshihisa (A)

Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.

Yasuchika Takeishi (Y)

Department of Cardiovascular Medicine, Fukushima Medical University, Fukushima, Japan.

Michael Egstrup (M)

Department of Cardiology, Bispebjerg University Hospital, København, Denmark.

Ida Gustafsson (I)

Department of Cardiology, Bispebjerg University Hospital, København, Denmark.

Hanna K Gaggin (HK)

Massachusetts General Hospital and Baim Institute for Clinical Research, Boston, USA.

Kai M Eggers (KM)

Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden.

Kurt Huber (K)

Faculty of Internal Medicine, Wilhelminenspital and Sigmund Freud University Medical School, Vienna, Austria.

Ioannis Tentzeris (I)

Faculty of Internal Medicine, Wilhelminenspital and Sigmund Freud University Medical School, Vienna, Austria.

Andrea Ripoli (A)

Fondazione Toscana G. Monasterio, Pisa, Italy.

Claudio Passino (C)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.
Fondazione Toscana G. Monasterio, Pisa, Italy.

Sandra Sanders-van Wijk (S)

Department of Cardiology, Maastricht University Medical Centre, PO Box 5800, 6202AZ, Maastricht, The Netherlands.

Michele Emdin (M)

Institute of Life Sciences, Scuola Superiore Sant'Anna, Pisa, Italy.
Fondazione Toscana G. Monasterio, Pisa, Italy.

Hans-Peter Brunner-La Rocca (HP)

Department of Cardiology, Maastricht University Medical Centre, PO Box 5800, 6202AZ, Maastricht, The Netherlands. hp.brunnerlarocca@mumc.nl.

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Classifications MeSH