Clinical and echocardiographic predictors of decompensation in acute severe aortic regurgitation due to infective endocarditis.


Journal

Echocardiography (Mount Kisco, N.Y.)
ISSN: 1540-8175
Titre abrégé: Echocardiography
Pays: United States
ID NLM: 8511187

Informations de publication

Date de publication:
04 2021
Historique:
revised: 24 02 2021
received: 27 12 2020
accepted: 28 02 2021
pubmed: 13 3 2021
medline: 10 7 2021
entrez: 12 3 2021
Statut: ppublish

Résumé

Patients with acute severe aortic regurgitation (AR) due to infective endocarditis can progress rapidly from the hemodynamically stable patient to pulmonary edema and cardiogenic shock. We sought to identify patients at risk of decompensation where emergent surgery should be undertaken. We identified 90 patients with acute severe AR from the echocardiography laboratory database. Baseline clinical, hemodynamic (heart rate (HR) and blood pressure (BP)), and echocardiographic data including mitral filling, premature mitral valve closure (PMVC), and diastolic mitral regurgitation (DMR) were identified. The primary endpoint was subsequent development of pulmonary edema or severe hemodynamic instability. Patients who met the primary endpoint had a higher HR (98.5 bpm vs 80.5 bpm), lower diastolic BP (54 mm Hg vs 61.5 mm Hg), higher mitral E-wave velocity (113 cm/s vs 83 cm/s), higher E/e' ratio (12.4 vs 8), higher proportion of DMR (27.8% vs 7.4%), and PMVC (25% vs 9.3%) than patients who did not meet the endpoint. The proportion of patients with the primary endpoint increased as HR increased ((≤81 bpm) 3/30 (10%), (81-94 bpm) 11/31 (35.5%), (≥94 bpm) 22/29 (75.9%), P < .0001) and as the diastolic BP reduced ((≤54 mm Hg) 19/31 (61.3%), (54-63 mm Hg) 12/31 (38.7%), (≥63 mm Hg) 5/28 (17.9%), P = .003). Independent predictors were a higher HR (OR 1.08 (95% CI 1.04-1.13) P = .0003) and DMR (OR 4.71 (95% CI 1.23-18.09), P = .02). Decompensation in acute severe AR is common. Independent predictors of decompensation are increasing HR(≥94 bpm) and the presence of DMR. Those with these adverse markers should be considered for emergent surgery.

Sections du résumé

BACKGROUND
Patients with acute severe aortic regurgitation (AR) due to infective endocarditis can progress rapidly from the hemodynamically stable patient to pulmonary edema and cardiogenic shock. We sought to identify patients at risk of decompensation where emergent surgery should be undertaken.
METHODS
We identified 90 patients with acute severe AR from the echocardiography laboratory database. Baseline clinical, hemodynamic (heart rate (HR) and blood pressure (BP)), and echocardiographic data including mitral filling, premature mitral valve closure (PMVC), and diastolic mitral regurgitation (DMR) were identified. The primary endpoint was subsequent development of pulmonary edema or severe hemodynamic instability.
RESULTS
Patients who met the primary endpoint had a higher HR (98.5 bpm vs 80.5 bpm), lower diastolic BP (54 mm Hg vs 61.5 mm Hg), higher mitral E-wave velocity (113 cm/s vs 83 cm/s), higher E/e' ratio (12.4 vs 8), higher proportion of DMR (27.8% vs 7.4%), and PMVC (25% vs 9.3%) than patients who did not meet the endpoint. The proportion of patients with the primary endpoint increased as HR increased ((≤81 bpm) 3/30 (10%), (81-94 bpm) 11/31 (35.5%), (≥94 bpm) 22/29 (75.9%), P < .0001) and as the diastolic BP reduced ((≤54 mm Hg) 19/31 (61.3%), (54-63 mm Hg) 12/31 (38.7%), (≥63 mm Hg) 5/28 (17.9%), P = .003). Independent predictors were a higher HR (OR 1.08 (95% CI 1.04-1.13) P = .0003) and DMR (OR 4.71 (95% CI 1.23-18.09), P = .02).
CONCLUSION
Decompensation in acute severe AR is common. Independent predictors of decompensation are increasing HR(≥94 bpm) and the presence of DMR. Those with these adverse markers should be considered for emergent surgery.

Identifiants

pubmed: 33711172
doi: 10.1111/echo.15028
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

590-595

Subventions

Organisme : Medical Research Council
ID : MR/R017468/1
Pays : United Kingdom

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

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Auteurs

Athina Chasapi (A)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.

Kamatamu A Mbonye (KA)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.

Omotomilola Bajomo (O)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.

William J Young (WJ)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.

Christopher Primus (C)

Department of Cardiology, St Bartholomew's Hospital, London, UK.

Shirish Ambekar (S)

Cardiovascular Surgery, St Bartholomew's Hospital, London, UK.

Kit Wong (K)

Cardiovascular Surgery, St Bartholomew's Hospital, London, UK.

Rakesh Uppal (R)

Cardiovascular Surgery, St Bartholomew's Hospital, London, UK.

Lewis Ceri Davies (LC)

Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.

Mohammed Y Khanji (MY)

Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.

Simon Woldman (S)

Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.
Institute of Cardiovascular Science, UCL, London, UK.

Guy Lloyd (G)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.
Institute of Cardiovascular Science, UCL, London, UK.

Sanjeev Bhattacharyya (S)

Echocardiography Laboratory, St Bartholomew's Hospital, London, UK.
Department of Cardiology, St Bartholomew's Hospital, London, UK.
William Harvey Research Institute, Queen Mary University of London, London, UK.
Institute of Cardiovascular Science, UCL, London, UK.

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