Changes in Default-Mode Network Associated With Childhood Trauma in Schizophrenia.


Journal

Schizophrenia bulletin
ISSN: 1745-1701
Titre abrégé: Schizophr Bull
Pays: United States
ID NLM: 0236760

Informations de publication

Date de publication:
21 08 2021
Historique:
pubmed: 7 4 2021
medline: 10 2 2022
entrez: 6 4 2021
Statut: ppublish

Résumé

There is considerable evidence of dysconnectivity within the default-mode network (DMN) in schizophrenia, as measured during resting-state functional MRI (rs-fMRI). History of childhood trauma (CT) is observed at a higher frequency in schizophrenia than in the general population, but its relationship to DMN functional connectivity has yet to be investigated. CT history and rs-fMRI data were collected in 65 individuals with schizophrenia and 132 healthy controls. Seed-based functional connectivity between each of 4 a priori defined seeds of the DMN (medial prefrontal cortex, right and left lateral parietal lobes, and the posterior cingulate cortex) and all other voxels of the brain were compared across groups. Effects of CT on functional connectivity were examined using multiple regression analyses. Where significant associations were observed, regression analyses were further used to determine whether variance in behavioral measures of Theory of Mind (ToM), previously associated with DMN recruitment, were explained by these associations. Seed-based analyses revealed evidence of widespread reductions in functional connectivity in patients vs controls, including between the left/right parietal lobe (LP) and multiple other regions, including the parietal operculum bilaterally. Across all subjects, increased CT scores were associated with reduced prefrontal-parietal connectivity and, in patients, with increased prefrontal-cerebellar connectivity also. These CT-associated differences in DMN connectivity also predicted variation in behavioral measures of ToM. These findings suggest that CT history is associated with variation in DMN connectivity during rs-fMRI in patients with schizophrenia and healthy participants, which may partly mediate associations observed between early life adversity and cognitive performance.

Sections du résumé

BACKGROUND
There is considerable evidence of dysconnectivity within the default-mode network (DMN) in schizophrenia, as measured during resting-state functional MRI (rs-fMRI). History of childhood trauma (CT) is observed at a higher frequency in schizophrenia than in the general population, but its relationship to DMN functional connectivity has yet to be investigated.
METHODS
CT history and rs-fMRI data were collected in 65 individuals with schizophrenia and 132 healthy controls. Seed-based functional connectivity between each of 4 a priori defined seeds of the DMN (medial prefrontal cortex, right and left lateral parietal lobes, and the posterior cingulate cortex) and all other voxels of the brain were compared across groups. Effects of CT on functional connectivity were examined using multiple regression analyses. Where significant associations were observed, regression analyses were further used to determine whether variance in behavioral measures of Theory of Mind (ToM), previously associated with DMN recruitment, were explained by these associations.
RESULTS
Seed-based analyses revealed evidence of widespread reductions in functional connectivity in patients vs controls, including between the left/right parietal lobe (LP) and multiple other regions, including the parietal operculum bilaterally. Across all subjects, increased CT scores were associated with reduced prefrontal-parietal connectivity and, in patients, with increased prefrontal-cerebellar connectivity also. These CT-associated differences in DMN connectivity also predicted variation in behavioral measures of ToM.
CONCLUSIONS
These findings suggest that CT history is associated with variation in DMN connectivity during rs-fMRI in patients with schizophrenia and healthy participants, which may partly mediate associations observed between early life adversity and cognitive performance.

Identifiants

pubmed: 33823040
pii: 6210462
doi: 10.1093/schbul/sbab025
pmc: PMC8379545
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1482-1494

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press on behalf of the Maryland Psychiatric Research Center.

Auteurs

Maria R Dauvermann (MR)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
McGovern Institute for Brain Research, Massachusetts Institute of Technology, Cambridge, MA.

David Mothersill (D)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
Department of Psychology, National College of Ireland, Dublin, Ireland.

Karolina I Rokita (KI)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.

Sinead King (S)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.

Laurena Holleran (L)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.

Ruan Kane (R)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.

Declan P McKernan (DP)

Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
Pharmacology and Therapeutics, National University of Ireland Galway, Galway, Ireland.

John P Kelly (JP)

Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
Pharmacology and Therapeutics, National University of Ireland Galway, Galway, Ireland.

Derek W Morris (DW)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
School of Natural Sciences, National University of Ireland Galway, Galway, Ireland.

Aiden Corvin (A)

Department of Psychiatry, Trinity Centre for Health Sciences, St. James's Hospital, Dublin, Ireland.

Brian Hallahan (B)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
Department of Psychiatry, Clinical Science Institute, National University of Ireland Galway, Galway, Ireland.

Colm McDonald (C)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.
Department of Psychiatry, Clinical Science Institute, National University of Ireland Galway, Galway, Ireland.

Gary Donohoe (G)

School of Psychology, National University of Ireland Galway, Galway, Ireland.
Center for Neuroimaging, Genetics and Cognition (NICOG), National University of Ireland Galway, Galway, Ireland.

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