Phase 1b/2 trial of tepotinib in sorafenib pretreated advanced hepatocellular carcinoma with MET overexpression.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
07 2021
Historique:
received: 02 10 2020
accepted: 23 02 2021
revised: 05 02 2021
pubmed: 8 4 2021
medline: 15 12 2021
entrez: 7 4 2021
Statut: ppublish

Résumé

This Phase 1b/2 study evaluated tepotinib, a highly selective MET inhibitor, in US/European patients with sorafenib pretreated advanced hepatocellular carcinoma (aHCC) with MET overexpression. Eligible adults had aHCC, progression after ≥4 weeks of sorafenib, and, for Phase 2 only, MET overexpression. Tepotinib was administered once daily at 300 or 500 mg in Phase 1b ('3 + 3' design), and at the recommended Phase 2 dose (RP2D) in Phase 2. Primary endpoints were dose-liming toxicities (DLTs; Phase 1b) and 12-week investigator-assessed progression-free survival (PFS; Phase 2). In Phase 1b (n = 17), no DLTs occurred and the RP2D was confirmed as 500 mg. In Phase 2 (n = 49), the primary endpoint was met: 12-week PFS was 63.3% (90% CI: 50.5-74.7), which was significantly greater than the predefined null hypothesis of ≤15% (one-sided binomial exact test: P < 0.0001). Median time to progression was 4 months. In Phase 2, 28.6% of patients had treatment-related Grade ≥3 adverse events, including peripheral oedema and lipase increase (both 6.1%). Tepotinib was generally well tolerated and the RP2D (500 mg) showed promising efficacy and, therefore, a positive benefit-risk balance in sorafenib pretreated aHCC with MET overexpression. ClinicalTrials.gov: NCT02115373.

Sections du résumé

BACKGROUND
This Phase 1b/2 study evaluated tepotinib, a highly selective MET inhibitor, in US/European patients with sorafenib pretreated advanced hepatocellular carcinoma (aHCC) with MET overexpression.
METHODS
Eligible adults had aHCC, progression after ≥4 weeks of sorafenib, and, for Phase 2 only, MET overexpression. Tepotinib was administered once daily at 300 or 500 mg in Phase 1b ('3 + 3' design), and at the recommended Phase 2 dose (RP2D) in Phase 2. Primary endpoints were dose-liming toxicities (DLTs; Phase 1b) and 12-week investigator-assessed progression-free survival (PFS; Phase 2).
RESULTS
In Phase 1b (n = 17), no DLTs occurred and the RP2D was confirmed as 500 mg. In Phase 2 (n = 49), the primary endpoint was met: 12-week PFS was 63.3% (90% CI: 50.5-74.7), which was significantly greater than the predefined null hypothesis of ≤15% (one-sided binomial exact test: P < 0.0001). Median time to progression was 4 months. In Phase 2, 28.6% of patients had treatment-related Grade ≥3 adverse events, including peripheral oedema and lipase increase (both 6.1%).
CONCLUSIONS
Tepotinib was generally well tolerated and the RP2D (500 mg) showed promising efficacy and, therefore, a positive benefit-risk balance in sorafenib pretreated aHCC with MET overexpression.
TRIAL REGISTRATION
ClinicalTrials.gov: NCT02115373.

Identifiants

pubmed: 33824476
doi: 10.1038/s41416-021-01334-9
pii: 10.1038/s41416-021-01334-9
pmc: PMC8292404
doi:

Substances chimiques

Antineoplastic Agents 0
Piperidines 0
Pyridazines 0
Pyrimidines 0
tepotinib 1IJV77EI07
Sorafenib 9ZOQ3TZI87
MET protein, human EC 2.7.10.1
Proto-Oncogene Proteins c-met EC 2.7.10.1

Banques de données

ClinicalTrials.gov
['NCT02115373']

Types de publication

Clinical Trial, Phase I Clinical Trial, Phase II Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

190-199

Commentaires et corrections

Type : ErratumIn

Informations de copyright

© 2021. The Author(s).

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Auteurs

Thomas Decaens (T)

University Grenoble Alpes, Department of Hepato-Gastroenterology and Digestive Oncology, CHU Grenoble Alpes, Institute for Advanced Biosciences INSERM U1209, Grenoble, France. tdecaens@chu-grenoble.fr.

Carlo Barone (C)

Medical Oncology, Policlinico Universitario A. Gemelli, Roma, Italy.

Eric Assenat (E)

Medical Oncology, CHU Saint Eloi, Montpellier, France.

Martin Wermke (M)

NCT/UCC Early Clinical Trial Unit, University Hospital Carl-Gustav-Carus, Dresden, Germany.

Angelica Fasolo (A)

Oncology, IRCCS San Raffaele, Milan, Italy.

Philippe Merle (P)

Service d'Hépato-Gastro-Entérologie, Hôpital de la Croix Rousse, Lyon, France.

Jean-Frédéric Blanc (JF)

Service d'Hépato-Gastroentérologie et d'Oncologie Digestive, Groupe Hospitalier Haut-Lévêque, CHU Bordeaux, Pessac, France.

Véronique Grando (V)

Service Hépatologie, Hôpital Jean-Verdier, Bondy, France.

Angelo Iacobellis (A)

Reparto di Gastroenterologia ed Endoscopia Digestiva, Ospedale Casa Sollievo della Sofferenza IRCCS, San Giovanni Rotondo, Italy.

Erica Villa (E)

Division of Gastroenterology Policlinico di Modena, Modena, Italy.

Joerg Trojan (J)

Gastrointestinal Oncology, Goethe University Hospital, Frankfurt, Germany.

Josef Straub (J)

Clinical Biomarker & Companion Diagnostics, Merck KGaA, Darmstadt, Germany.

Rolf Bruns (R)

Biostatistics, Merck KGaA, Darmstadt, Germany.

Karin Berghoff (K)

Global Patient Safety Innovation, Merck KGaA, Darmstadt, Germany.

Juergen Scheele (J)

Global Clinical Development Oncology, Merck KGaA, Darmstadt, Germany.

Eric Raymond (E)

Medical Oncology, Paris-St Joseph Hospital, Paris, France.

Sandrine Faivre (S)

Medical Oncology, Saint-Louis Hospital & Paris 7 University, Paris, France.

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