Deletion of the Imprinted
Phlda2
fetal growth
genomic imprinting
passive permeability
placenta
Journal
Genes
ISSN: 2073-4425
Titre abrégé: Genes (Basel)
Pays: Switzerland
ID NLM: 101551097
Informations de publication
Date de publication:
25 04 2021
25 04 2021
Historique:
received:
14
03
2021
revised:
09
04
2021
accepted:
23
04
2021
entrez:
30
4
2021
pubmed:
1
5
2021
medline:
24
8
2021
Statut:
epublish
Résumé
Genomic imprinting, an epigenetic phenomenon that causes the expression of a small set of genes in a parent-of-origin-specific manner, is thought to have co-evolved with placentation. Many imprinted genes are expressed in the placenta, where they play diverse roles related to development and nutrient supply function. However, only a small number of imprinted genes have been functionally tested for a role in nutrient transfer capacity in relation to the structural characteristics of the exchange labyrinthine zone. Here, we examine the transfer capacity in a mouse model deficient for the maternally expressed
Identifiants
pubmed: 33922969
pii: genes12050639
doi: 10.3390/genes12050639
pmc: PMC8146920
pii:
doi:
Substances chimiques
Nuclear Proteins
0
TSSC3 protein
0
beta-Alanine
11P2JDE17B
3-O-Methylglucose
146-72-5
2,2-dimethyl-beta-alanine
19036-43-2
Mannitol
3OWL53L36A
Inulin
9005-80-5
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Biotechnology and Biological Sciences Research Council
ID : BB/B50118X/1
Pays : United Kingdom
Organisme : Biotechnology and Biological Sciences Research Council
ID : BB/F02293X/1
Pays : United Kingdom
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